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Plos One|May 3, 2011
Reversal of diabetic nephropathy by a ketogenic dietMichal M Poplawski, Jason W Mastaitis, Fumiko Isoda, et al.
Diabetes|October 28, 2003
Transgenic neuronal expression of proopiomelanocortin attenuates hyperphagic response to fasting and reverses metabolic impairments in leptin-deficient obese miceTooru M Mizuno, Kevin A Kelley, Giulio M Pasinetti, et al.
Brain Research|September 6, 2003
The fatty acid synthase inhibitor cerulenin and feeding, like leptin, activate hypothalamic pro-opiomelanocortin (POMC) neuronsI-Wei Shu, Daniel L Lindenberg, Tooru M Mizuno, et al.
Appetite|December 5, 2006
Low-carbohydrate diets cause obesity, low-carbohydrate diets reverse obesity: a metabolic mechanism resolving the paradoxCharles V Mobbs, Jason Mastaitis, Kelvin Yen, et al.
Frontiers in Neuroendocrinology|December 25, 2012
Role of the hypothalamus in mediating protective effects of dietary restriction during agingPenny A Dacks, Cesar L Moreno, Esther S Kim, et al.
Plos Biology|November 20, 2009
Role of CBP and SATB-1 in aging, dietary restriction, and insulin-like signalingMinhua Zhang, Michal Poplawski, Kelvin Yen, et al.
Plos One|November 11, 2016
Role of Hypothalamic Creb-Binding Protein in Obesity and Molecular Reprogramming of Metabolic SubstratesCesar L Moreno, Linda Yang, Penny A Dacks, et al.
Neurochemical Research|December 5, 2002
Validated genomic approach to study differentially expressed genes in complex tissuesElisa Wurmbach, Javier González-Maeso, Tony Yuen, et al.
Physiology & Behavior|June 1, 2005
Impaired glucose signaling as a cause of obesity and the metabolic syndrome: the glucoadipostatic hypothesisCharles V Mobbs, Fumiko Isoda, Hideo Makimura, et al.
Frontiers in Physiology|January 6, 2022
Serotonin and Dopamine Mimic Glucose-Induced Reinforcement in C. elegans: Potential Role of NSM Neurons and the Serotonin Subtype 4 ReceptorElizabeth K C Schwartz, Eitan N Sosner, Hayley E Desmond, et al.
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