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American Journal of Nephrology|July 3, 2013
GLUT1 regulation of the pro-sclerotic mediators of diabetic nephropathyCharles W Heilig, Dilip K Deb, Afu Abdul, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 16, 2003
Glucose transporter-1-deficient mice exhibit impaired development and deformities that are similar to diabetic embryopathyCharles W Heilig, Thomas Saunders, Frank C Brosius, et al.
Clinical Nephrology|September 10, 2019
Effect of race and ethnicity on renal transplant referralSarah T Suliman, Jeremy Carlson, Carmen Smotherman, et al.
Journal of the American Society of Nephrology : JASN|November 26, 2004
Mouse models of diabetic nephropathyMatthew D Breyer, Erwin Böttinger, Frank C Brosius, et al.
American Journal of Physiology. Renal Physiology|April 9, 2010
Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosisYouli Wang, Kathleen Heilig, Thomas Saunders, et al.
Advances in Chronic Kidney Disease|April 12, 2005
Diabetic nephropathy: of mice and menMatthew D Breyer, Erwin Böttinger, Frank C Brosius, et al.
Neurobiology of Disease|June 12, 2012
Glut1 deficiency (G1D): epilepsy and metabolic dysfunction in a mouse model of the most common human phenotypeIsaac Marin-Valencia, Levi B Good, Qian Ma, et al.
American Journal of Physiology. Renal Physiology|April 9, 2010
Podocyte-specific overexpression of GLUT1 surprisingly reduces mesangial matrix expansion in diabetic nephropathy in miceHongyu Zhang, MaryLee Schin, Jharna Saha, et al.
Kidney International|September 13, 2003
Regulation of cytosolic pH and lactic acid release in mesangial cells overexpressing GLUT1Karl S Lang, Matthias M Mueller, Valerie Tanneur, et al.
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