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Parkinson'S Disease|June 28, 2012
αSynuclein and Mitochondrial Dysfunction: A Pathogenic Partnership in Parkinson's Disease?David Protter, Charmaine Lang, Antony A CooperProgress in Brain Research|April 6, 2020
What we can learn from iPSC-derived cellular models of Parkinson's diseaseMaria Claudia Caiazza, Charmaine Lang, Richard Wade-MartinsHuman Molecular Genetics|January 19, 2017
Transcriptomic profiling of purified patient-derived dopamine neurons identifies convergent perturbations and therapeutics for Parkinson's diseaseCynthia Sandor, Paul Robertson, Charmaine Lang, et al.Cell Stem Cell|December 4, 2018
Single-Cell Sequencing of iPSC-Dopamine Neurons Reconstructs Disease Progression and Identifies HDAC4 as a Regulator of Parkinson Cell PhenotypesCharmaine Lang, Kieran R Campbell, Brent J Ryan, et al.NPJ Parkinson'S Disease|April 12, 2024
Early deficits in an in vitro striatal microcircuit model carrying the Parkinson's GBA-N370S mutationQuyen B Do, Humaira Noor, Ricardo Marquez-Gomez, et al.Human Molecular Genetics|February 13, 2019
Cellular α-synuclein pathology is associated with bioenergetic dysfunction in Parkinson's iPSC-derived dopamine neuronsFederico Zambon, Marta Cherubini, Hugo J R Fernandes, et al.Stem Cell Reports|February 25, 2016
ER Stress and Autophagic Perturbations Lead to Elevated Extracellular α-Synuclein in GBA-N370S Parkinson's iPSC-Derived Dopamine NeuronsHugo J R Fernandes, Elizabeth M Hartfield, Helen C Christian, et al.Pageof 1