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Biorxiv : the Preprint Server for Biology|April 10, 2026
c-MYC is an aggregation-prone, amyloidogenic proteinLing Lin, Kun-Han Chuang, Chengkai DaiScience Advances|November 12, 2020
HSF1 physically neutralizes amyloid oligomers to empower overgrowth and bestow neuroprotectionZijian Tang, Kuo-Hui Su, Meng Xu, et al.Neoplasia (New York, N.Y.)|April 2, 2002
MYC expression promotes the proliferation of neural progenitor cells in culture and in vivoDan Fults, Carolyn Pedone, Chengkai Dai, et al.Cell|September 25, 2007
Heat shock factor 1 is a powerful multifaceted modifier of carcinogenesisChengkai Dai, Luke Whitesell, Arlin B Rogers, et al.Molecular Cell|September 29, 2019
Heat Shock Factor 1 Is a Direct Antagonist of AMP-Activated Protein KinaseKuo-Hui Su, Siyuan Dai, Zijian Tang, et al.Biorxiv : the Preprint Server for Biology|March 16, 2026
Driving proteomic imbalance in malignancy provokes proteomic catastrophe and confers tumor suppressionBabul Moni Ram, Omprakash Shriwas, Meng Xu, et al.The EMBO Journal|November 27, 2014
Suppression of the HSF1-mediated proteotoxic stress response by the metabolic stress sensor AMPKSiyuan Dai, Zijian Tang, Junyue Cao, et al.Neuron Glia Biology|October 19, 2006
Overexpression of c-MYC promotes an undifferentiated phenotype in cultured astrocytes and allows elevated Ras and Akt signaling to induce gliomas from GFAP-expressing cells in miceAndrew B Lassman, Chengkai Dai, Gregory N Fuller, et al.Cancer Research|July 17, 2004
Dose-dependent effects of platelet-derived growth factor-B on glial tumorigenesisAlan H Shih, Chengkai Dai, Xiaoyi Hu, et al.Neoplasia (New York, N.Y.)|June 22, 2005
The characteristics of astrocytomas and oligodendrogliomas are caused by two distinct and interchangeable signaling formatsChengkai Dai, Yelena Lyustikman, Alan Shih, et al.Pageof 3