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The Journal of Clinical Investigation|March 18, 2020
Sel1L-Hrd1 ER-associated degradation maintains β cell identity via TGF-β signalingNeha Shrestha, Tongyu Liu, Yewei Ji, et al.
Blood Advances|February 28, 2018
Inhibition of the IRE-1α/XBP-1 pathway prevents chronic GVHD and preserves the GVL effect in miceSteven D Schutt, Yongxia Wu, Chih-Hang Anthony Tang, et al.
Nature Cell Biology|May 2, 2017
Regulated IRE1-dependent mRNA decay sets the threshold for dendritic cell survivalSimon J Tavernier, Fabiola Osorio, Lana Vandersarren, et al.
Blood|June 14, 2012
Overexpression of TCL1 activates the endoplasmic reticulum stress response: a novel mechanism of leukemic progression in miceCrystina L Kriss, Javier A Pinilla-Ibarz, Adam W Mailloux, et al.
The Journal of Biological Chemistry|May 17, 2021
An interdomain helix in IRE1α mediates the conformational change required for the sensor's activationDaniela Ricci, Stephen Tutton, Ilaria Marrocco, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
Targeting Endothelial PERK Accelerates Lymphoid Regeneration by Enhancing DLL4-NOTCH3 Signaling at the Pre-B NicheBingqing Zou, Qiuyun Chen, Junjun Zheng, et al.
Nature Cell Biology|May 4, 2023
SEL1L-HRD1 endoplasmic reticulum-associated degradation controls STING-mediated innate immunity by limiting the size of the activable STING poolYewei Ji, Yuan Luo, Yating Wu, et al.
Molecular Biology of the Cell|October 3, 2019
Mitochondrial lipid droplet formation as a detoxification mechanism to sequester and degrade excessive urothelial membranesYi Liao, Daniel K L Tham, Feng-Xia Liang, et al.
The Journal of Clinical Investigation|February 16, 2021
Endoplasmic reticulum-associated degradation is required for nephrin maturation and kidney glomerular filtration functionSei Yoshida, Xiaoqiong Wei, Gensheng Zhang, et al.
The Journal of Clinical Investigation|July 6, 2021
Distinct mechanisms govern populations of myeloid-derived suppressor cells in chronic viral infection and cancerEvgenii N Tcyganov, Shino Hanabuchi, Ayumi Hashimoto, et al.
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