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Cancer Research|April 7, 2019
Long Noncoding RNA ELIT-1 Acts as a Smad3 Cofactor to Facilitate TGFβ/Smad Signaling and Promote Epithelial-Mesenchymal TransitionSatoshi Sakai, Tatsuya Ohhata, Kyoko Kitagawa, et al.
Biochimica Et Biophysica Acta. Molecular Cell Research|August 8, 2022
Chromatin-remodeling factor BAZ1A/ACF1 targets UV damage sites in an MLL1-dependent manner to facilitate nucleotide excision repairTakafumi Koyauchi, Hiroyuki Niida, Akira Motegi, et al.
Biochemical and Biophysical Research Communications|December 14, 2005
Mdm2-mediated pRB downregulation is involved in carcinogenesis in a p53-independent mannerSeiichi Miwa, Chiharu Uchida, Kyoko Kitagawa, et al.
Kidney International|December 17, 2003
Treatment with anti-TGF-beta antibody ameliorates chronic progressive nephritis by inhibiting Smad/TGF-beta signalingHirotaka Fukasawa, Tatsuo Yamamoto, Hiroyuki Suzuki, et al.
Cancer Science|May 16, 2009
High expression of Pirh2, an E3 ligase for p27, is associated with low expression of p27 and poor prognosis in head and neck cancersMai Shimada, Kyoko Kitagawa, Yoh Dobashi, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 3, 2004
Down-regulation of Smad7 expression by ubiquitin-dependent degradation contributes to renal fibrosis in obstructive nephropathy in miceHirotaka Fukasawa, Tatsuo Yamamoto, Akashi Togawa, et al.
The EMBO Journal|December 4, 2004
Enhanced Mdm2 activity inhibits pRB function via ubiquitin-dependent degradationChiharu Uchida, Seiichi Miwa, Kyoko Kitagawa, et al.
Molecular Cancer Research : MCR|June 22, 2017
Homeobox Transcription Factor NKX2-1 Promotes Cyclin D1 Transcription in Lung AdenocarcinomasMasanori Harada, Satoshi Sakai, Tatsuya Ohhata, et al.
Cancer Research|September 5, 2006
Degradation of Tob1 mediated by SCFSkp2-dependent ubiquitinationYoshihiro Hiramatsu, Kyoko Kitagawa, Toru Suzuki, et al.
Molecular and Cellular Biology|November 18, 2015
UV Damage-Induced Phosphorylation of HBO1 Triggers CRL4DDB2-Mediated Degradation To Regulate Cell ProliferationRyoichi Matsunuma, Hiroyuki Niida, Tatsuya Ohhata, et al.
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