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Kidney International|May 17, 2016
Loss of Glis2/NPHP7 causes kidney epithelial cell senescence and suppresses cyst growth in the Kif3a mouse model of cystic kidney diseaseDongmei Lu, Alysha Rauhauser, Binghua Li, et al.
Cell Reports|July 25, 2025
Cerebellar contributions to tau-mediated cognitive and behavioral dysfunctionLidiette Angeles-Perez, Chongyu Ren, Juliana Neniel, et al.
American Journal of Physiology. Renal Physiology|August 21, 2015
Hedgehog signaling indirectly affects tubular cell survival after obstructive kidney injuryAlysha A Rauhauser, Chongyu Ren, Dongmei Lu, et al.
Iscience|May 23, 2022
Increased glycine contributes to synaptic dysfunction and early mortality in Nprl2 seizure modelBrianne Dentel, Lidiette Angeles-Perez, Chongyu Ren, et al.
JCI Insight|September 20, 2019
Disruption of Robo2-Baiap2 integrated signaling drives cystic diseaseQinggang Li, Shaoyuan Cui, Qian Ma, et al.
The American Journal of Pathology|November 3, 2019
Innate Immune Signaling Contributes to Tubular Cell Senescence in the Glis2 Knockout Mouse Model of NephronophthisisHeng Jin, Yan Zhang, Dingxiao Liu, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 23, 2010
Knockout of Na+/Ca2+ exchanger in smooth muscle attenuates vasoconstriction and L-type Ca2+ channel current and lowers blood pressureJin Zhang, Chongyu Ren, Ling Chen, et al.
JCI Insight|January 25, 2019
Epithelial innate immunity mediates tubular cell senescence after kidney injuryHeng Jin, Yan Zhang, Qiong Ding, et al.
American Journal of Physiology. Renal Physiology|February 19, 2016
Loss of diacylglycerol kinase epsilon in mice causes endothelial distress and impairs glomerular Cox-2 and PGE2 productionJili Zhu, Moumita Chaki, Dongmei Lu, et al.
Nature Neuroscience|July 15, 2020
Regulation of autism-relevant behaviors by cerebellar-prefrontal cortical circuitsElyza Kelly, Fantao Meng, Hirofumi Fujita, et al.
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