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Chrishan S Samuel

Showing results (61-70 of 173) with videos related to

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Nature Reviews. Cardiology|November 26, 2009
Cardiovascular effects of relaxin: from basic science to clinical therapyXiao-Jun Du, Ross A D Bathgate, Chrishan S Samuel, et al.
Kidney International|May 20, 2004
Relaxin-1-deficient mice develop an age-related progression of renal fibrosisChrishan S Samuel, Chongxin Zhao, Courtney P Bond, et al.
Stem Cell Research|October 2, 2015
Mesenchymal stem cells and serelaxin synergistically abrogate established airway fibrosis in an experimental model of chronic allergic airways diseaseSimon G Royce, Matthew Shen, Krupesh P Patel, et al.
The American Journal of Pathology|June 6, 2009
Human umbilical cord mesenchymal stem cells reduce fibrosis of bleomycin-induced lung injuryYuben Moodley, Daniel Atienza, Ursula Manuelpillai, et al.
Frontiers in Pharmacology|June 15, 2017
Epigenetic Modifications to H3K9 in Renal Tubulointerstitial Cells after Unilateral Ureteric Obstruction and TGF-β1 StimulationTimothy D Hewitson, Stephen G Holt, Sven-Jean Tan, et al.
Cardiovascular Research|February 5, 2003
Increased myocardial collagen and ventricular diastolic dysfunction in relaxin deficient mice: a gender-specific phenotypeXiao-Jun Du, Chrishan S Samuel, Xiao-Ming Gao, et al.
Journal of Immunology Research|December 10, 2019
Distinct Redox Signalling following Macrophage Activation Influences Profibrotic ActivityCaitlin V Lewis, Antony Vinh, Henry Diep, et al.
The Journal of Investigative Dermatology|September 28, 2005
The relaxin gene knockout mouse: a model of progressive sclerodermaChrishan S Samuel, Chongxin Zhao, Qing Yang, et al.
Hypertension (Dallas, Tex. : 1979)|May 29, 2014
Serelaxin is a more efficacious antifibrotic than enalapril in an experimental model of heart diseaseChrishan S Samuel, Hasangika Bodaragama, Jacqueline Y Chew, et al.
Frontiers in Pharmacology|April 12, 2016
The Anti-fibrotic Actions of Relaxin Are Mediated Through a NO-sGC-cGMP-Dependent Pathway in Renal Myofibroblasts In Vitro and Enhanced by the NO Donor, Diethylamine NONOateChao Wang, Barbara K Kemp-Harper, Martina Kocan, et al.
Pageof 18

Showing results (61-70 of 173) with videos related to

Sort By:
Pageof 18
Nature Reviews. Cardiology|November 26, 2009
Cardiovascular effects of relaxin: from basic science to clinical therapyXiao-Jun Du, Ross A D Bathgate, Chrishan S Samuel, et al.
Kidney International|May 20, 2004
Relaxin-1-deficient mice develop an age-related progression of renal fibrosisChrishan S Samuel, Chongxin Zhao, Courtney P Bond, et al.
Stem Cell Research|October 2, 2015
Mesenchymal stem cells and serelaxin synergistically abrogate established airway fibrosis in an experimental model of chronic allergic airways diseaseSimon G Royce, Matthew Shen, Krupesh P Patel, et al.
The American Journal of Pathology|June 6, 2009
Human umbilical cord mesenchymal stem cells reduce fibrosis of bleomycin-induced lung injuryYuben Moodley, Daniel Atienza, Ursula Manuelpillai, et al.
Frontiers in Pharmacology|June 15, 2017
Epigenetic Modifications to H3K9 in Renal Tubulointerstitial Cells after Unilateral Ureteric Obstruction and TGF-β1 StimulationTimothy D Hewitson, Stephen G Holt, Sven-Jean Tan, et al.
Cardiovascular Research|February 5, 2003
Increased myocardial collagen and ventricular diastolic dysfunction in relaxin deficient mice: a gender-specific phenotypeXiao-Jun Du, Chrishan S Samuel, Xiao-Ming Gao, et al.
Journal of Immunology Research|December 10, 2019
Distinct Redox Signalling following Macrophage Activation Influences Profibrotic ActivityCaitlin V Lewis, Antony Vinh, Henry Diep, et al.
The Journal of Investigative Dermatology|September 28, 2005
The relaxin gene knockout mouse: a model of progressive sclerodermaChrishan S Samuel, Chongxin Zhao, Qing Yang, et al.
Hypertension (Dallas, Tex. : 1979)|May 29, 2014
Serelaxin is a more efficacious antifibrotic than enalapril in an experimental model of heart diseaseChrishan S Samuel, Hasangika Bodaragama, Jacqueline Y Chew, et al.
Frontiers in Pharmacology|April 12, 2016
The Anti-fibrotic Actions of Relaxin Are Mediated Through a NO-sGC-cGMP-Dependent Pathway in Renal Myofibroblasts In Vitro and Enhanced by the NO Donor, Diethylamine NONOateChao Wang, Barbara K Kemp-Harper, Martina Kocan, et al.
Pageof 18