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Physiological Genomics|January 25, 2007
Induction of dendritic cell-like phenotype in macrophages during foam cell formationHyung Jun Cho, Pavel Shashkin, Christian A Gleissner, et al.Innate Immunity|February 14, 2016
Differential regulation of aldose reductase expression during macrophage polarization depends on hyperglycemiaChristian Erbel, Gregor Rupp, Gabriele Domschke, et al.Journal of Visualized Experiments : Jove|June 25, 2013
An in vitro model to study heterogeneity of human macrophage differentiation and polarizationChristian Erbel, Gregor Rupp, Christian M Helmes, et al.Clinical Transplantation|June 5, 2003
Diagnostic efficiency of rejection monitoring after heart transplantation with cardiac troponin T is improved in specific patient subgroupsChristian A Gleissner, Roland Klingenberg, Winfried Nottmeyer, et al.Cardiovascular Research|August 3, 2019
Functional association of a CD40 gene single-nucleotide polymorphism with the pathogenesis of coronary heart diseaseCheryl S Sultan, Michael Weitnauer, Martin Turinsky, et al.Transplantation|March 17, 2004
Reduced hemoglobin after heart transplantation is no independent risk factor for survival but is associated closely with impaired renal functionChristian A Gleissner, Ammanouil Murat, Susanne Schäfer, et al.International Journal of Cardiology|April 2, 2015
Prevalence of M4 macrophages within human coronary atherosclerotic plaques is associated with features of plaque instabilityChristian Erbel, Antonia Wolf, Felix Lasitschka, et al.Drug Design, Development and Therapy|December 5, 2014
CCL19 and CCL21 modulate the inflammatory milieu in atherosclerotic lesionsMohammadreza Akhavanpoor, Christian A Gleissner, Stephanie Gorbatsch, et al.Drug Design, Development and Therapy|November 16, 2013
Heart rate reduction for 36 months with ivabradine reduces left ventricular mass in cardiac allograft recipients: a long-term follow-up studyAndreas O Doesch, Susanne Mueller, Christian Erbel, et al.Basic Research in Cardiology|December 1, 2010
Expression of IL-17A in human atherosclerotic lesions is associated with increased inflammation and plaque vulnerabilityChristian Erbel, Thomas J Dengler, Susanne Wangler, et al.Pageof 6