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Nephron. Experimental Nephrology|February 3, 2009
Thrombospondin in renal diseaseChristian Hugo, Christoph Daniel
Nephrology, Dialysis, Transplantation : Official Publication of the European Dialysis and Transplant Association - European Renal Association|July 4, 2008
PDE-5 inhibition impedes TSP-1 expression, TGF-beta activation and matrix accumulation in experimental glomerulonephritisBernd Hohenstein, Christoph Daniel, Sandra Wittmann, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|February 13, 2008
Nestin expression in repopulating mesangial cells promotes their proliferationChristoph Daniel, Heinz Albrecht, Andrea Lüdke, et al.
American Journal of Physiology. Renal Physiology|September 4, 2009
Thrombospondin-2 therapy ameliorates experimental glomerulonephritis via inhibition of cell proliferation, inflammation, and TGF-beta activationChristoph Daniel, Andrea Wagner, Bernd Hohenstein, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 6, 2004
The rapamycin derivative RAD inhibits mesangial cell migration through the CDK-inhibitor p27KIP1Christoph Daniel, Jeffrey Pippin, Stuart J Shankland, et al.
Transplantation|December 20, 2007
The mTOR inhibitor everolimus induces proteinuria and renal deterioration in the remnant kidney model in the ratRegina Vogelbacher, Sandra Wittmann, Andrea Braun, et al.
Diabetes|September 20, 2007
Thrombospondin-1 is an endogenous activator of TGF-beta in experimental diabetic nephropathy in vivoChristoph Daniel, Kathrin Schaub, Kerstin Amann, et al.
Journal of the American Society of Nephrology : JASN|February 9, 2007
Thrombospondin 2 functions as an endogenous regulator of angiogenesis and inflammation in experimental glomerulonephritis in miceChristoph Daniel, Kerstin Amann, Bernd Hohenstein, et al.
Plos One|December 31, 2013
Long-term gene therapy with thrombospondin 2 inhibits TGF-β activation, inflammation and angiogenesis in chronic allograft nephropathyChristoph Daniel, Regina Vogelbacher, Andrea Stief, et al.
American Journal of Physiology. Renal Physiology|November 25, 2004
Stimulation of soluble guanylyl cyclase inhibits mesangial cell proliferation and matrix accumulation in experimental glomerulonephritisBernd Hohenstein, Christoph Daniel, Andrea Wagner, et al.
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