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Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|August 30, 2019
Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition PoreElena C Castillo, José A Morales, Héctor Chapoy-Villanueva, et al.
Journal of Diabetes Research|September 23, 2020
Serum Irisin Levels, Endothelial Dysfunction, and Inflammation in Pediatric Patients with Type 2 Diabetes Mellitus and Metabolic SyndromeAnna S Huerta-Delgado, Daniel N Roffe-Vazquez, Adrian M Gonzalez-Gil, et al.
American Journal of Physiology. Heart and Circulatory Physiology|December 21, 2014
Impaired oxidative metabolism and calcium mishandling underlie cardiac dysfunction in a rat model of post-acute isoproterenol-induced cardiomyopathyB Cicero Willis, Ayleen Salazar-Cantú, Christian Silva-Platas, et al.
Oxidative Medicine and Cellular Longevity|December 5, 2017
Proinflammatory Cytokines Are Soluble Mediators Linked with Ventricular Arrhythmias and Contractile Dysfunction in a Rat Model of Metabolic SyndromeEvaristo Fernández-Sada, Alejandro Torres-Quintanilla, Christian Silva-Platas, et al.
Magnetic Resonance Imaging|July 16, 2022
Association of irisin levels with cardiac magnetic resonance, inflammatory, and biochemical parameters in patients with chronic heart failure versus controlsAnna S Huerta-Delgado, Daniel N Roffe-Vazquez, Eder Luna-Ceron, et al.
Journal of Bioenergetics and Biomembranes|January 8, 2016
Antineoplastic copper coordinated complexes (Casiopeinas) uncouple oxidative phosphorylation and induce mitochondrial permeability transition in cardiac mitochondria and cardiomyocytesChristian Silva-Platas, Carlos Enrique Guerrero-Beltrán, Mariana Carrancá, et al.
JACC. Basic to Translational Science|April 29, 2024
Mitochondrial Ca<sup>2+</sup> Uniporter-Dependent Energetic Dysfunction Drives Hypertrophy in Heart FailureHugo Alves-Figueiredo, Christian Silva-Platas, Manuel Estrada, et al.
British Journal of Pharmacology|November 15, 2025
Mitochondrial Ca<sup>2+</sup> overload is a pivotal risk factor for lethal ventricular arrhythmias due to the oxidation of mitochondrial respirasome and energetic failureFelipe de Jesús Salazar-Ramírez, Luis Alberto Luévano-Martínez, Abraham Méndez-Fernández, et al.
Particle and Fibre Toxicology|May 9, 2020
Amorphous SiO2 nanoparticles promote cardiac dysfunction via the opening of the mitochondrial permeability transition pore in rat heart and human cardiomyocytesOmar Lozano, Christian Silva-Platas, Héctor Chapoy-Villanueva, et al.
Oxidative Medicine and Cellular Longevity|May 17, 2018
Ex Vivo Cardiotoxicity of Antineoplastic Casiopeinas Is Mediated through Energetic Dysfunction and Triggered Mitochondrial-Dependent ApoptosisChristian Silva-Platas, César A Villegas, Yuriana Oropeza-Almazán, et al.
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