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BMC Genomics|March 19, 2013
The mutation rate of mycobacterial repetitive unit loci in strains of M. tuberculosis from cynomolgus macaque infectionMark N Ragheb, Christopher B Ford, Michael R Chase, et al.Nature Communications|April 10, 2019
Coordinated host-pathogen transcriptional dynamics revealed using sorted subpopulations and single macrophages infected with Candida albicansJosé F Muñoz, Toni Delorey, Christopher B Ford, et al.Plos Pathogens|November 13, 2012
Distinct effects on diversifying selection by two mechanisms of immunity against Streptococcus pneumoniaeYuan Li, Todd Gierahn, Claudette M Thompson, et al.Journal of Immunology (Baltimore, Md. : 1950)|December 17, 2014
Computational modeling predicts IL-10 control of lesion sterilization by balancing early host immunity-mediated antimicrobial responses with caseation during mycobacterium tuberculosis infectionNicholas A Cilfone, Christopher B Ford, Simeone Marino, et al.Msphere|October 26, 2018
Transcriptional Heterogeneity of <i>Cryptococcus gattii</i> VGII Compared with Non-VGII Lineages Underpins Key Pathogenicity PathwaysRhys A Farrer, Christopher B Ford, Johanna Rhodes, et al.Nature Medicine|December 17, 2013
Sterilization of granulomas is common in active and latent tuberculosis despite within-host variability in bacterial killingPhilana Ling Lin, Christopher B Ford, M Teresa Coleman, et al.Nature Genetics|June 11, 2013
Mycobacterium tuberculosis mutation rate estimates from different lineages predict substantial differences in the emergence of drug-resistant tuberculosisChristopher B Ford, Rupal R Shah, Midori Kato Maeda, et al.Nature Genetics|April 21, 2015
DNA replication fidelity in Mycobacterium tuberculosis is mediated by an ancestral prokaryotic proofreaderJeremy M Rock, Ulla F Lang, Michael R Chase, et al.Plos Medicine|March 10, 2020
Fecal microbiota transplantation for the improvement of metabolism in obesity: The FMT-TRIM double-blind placebo-controlled pilot trialElaine W Yu, Liu Gao, Petr Stastka, et al.Microbiology (Reading, England)|August 1, 2008
Expression of the Helicobacter pylori adhesin SabA is controlled via phase variation and the ArsRS signal transduction systemAndrew C Goodwin, Daniel M Weinberger, Christopher B Ford, et al.Pageof 3