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Neuropathology : Official Journal of the Japanese Society of Neuropathology|June 6, 2009
TDP-43 is consistently co-localized with ubiquitinated inclusions in sporadic and Guam amyotrophic lateral sclerosis but not in familial amyotrophic lateral sclerosis with and without SOD1 mutationsSatomi Maekawa, P Nigel Leigh, Andrew King, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|June 6, 2014
Autosomal dominant inheritance of rapidly progressive amyotrophic lateral sclerosis due to a truncation mutation in the fused in sarcoma (FUS) geneLouisa Kent, Thomas N Vizard, Bradley N Smith, et al.
Acta Neuropathologica|November 22, 2011
p62 positive, TDP-43 negative, neuronal cytoplasmic and intranuclear inclusions in the cerebellum and hippocampus define the pathology of C9orf72-linked FTLD and MND/ALSSafa Al-Sarraj, Andrew King, Claire Troakes, et al.
Brain : a Journal of Neurology|May 18, 2010
Nuclear import impairment causes cytoplasmic trans-activation response DNA-binding protein accumulation and is associated with frontotemporal lobar degenerationAgnes L Nishimura, Vera Zupunski, Claire Troakes, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|October 4, 2012
Is language impairment more common than executive dysfunction in amyotrophic lateral sclerosis?Lorna J Taylor, Richard G Brown, Stella Tsermentseli, et al.
Amyotrophic Lateral Sclerosis : Official Publication of the World Federation of Neurology Research Group on Motor Neuron Diseases|April 23, 2008
Association study on glutathione S-transferase omega 1 and 2 and familial ALSElsmarieke van de Giessen, Isabella Fogh, Sumana Gopinath, et al.
Brain : a Journal of Neurology|September 22, 2011
The risk to relatives of patients with sporadic amyotrophic lateral sclerosisMartha F Hanby, Kirsten M Scott, William Scotton, et al.
Amyotrophic Lateral Sclerosis & Frontotemporal Degeneration|May 28, 2014
Evidence of an environmental effect on survival in ALSNoa Keren, Kirsten M Scott, Miho Tsuda, et al.
Nature Communications|January 13, 2015
Human iPSC-derived motoneurons harbouring TARDBP or C9ORF72 ALS mutations are dysfunctional despite maintaining viabilityAnna-Claire Devlin, Karen Burr, Shyamanga Borooah, et al.
Plos One|December 10, 2009
The role of copy number variation in susceptibility to amyotrophic lateral sclerosis: genome-wide association study and comparison with published lociLouise V Wain, Inti Pedroso, John E Landers, et al.
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