Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Christopher J Holler

Showing results (1-10 of 12) with videos related to

Pageof 2
Sort By:
Eneuro|August 23, 2017
Intracellular Proteolysis of Progranulin Generates Stable, Lysosomal Granulins that Are Haploinsufficient in Patients with Frontotemporal Dementia Caused by <i>GRN</i> MutationsChristopher J Holler, Georgia Taylor, Qiudong Deng, et al.
Protein Science : a Publication of the Protein Society|June 14, 2017
Disulfide bonds and disorder in granulin-3: An unusual handshake between structural stability and plasticityGaurav Ghag, Christopher J Holler, Georgia Taylor, et al.
Journal of Neurochemistry|December 9, 2009
BACE1 and BACE2 enzymatic activities in Alzheimer's diseaseRachel R Ahmed, Christopher J Holler, Robin L Webb, et al.
Journal of Alzheimer'S Disease : JAD|July 16, 2014
Bridging integrator 1 (BIN1) protein expression increases in the Alzheimer's disease brain and correlates with neurofibrillary tangle pathologyChristopher J Holler, Paulina R Davis, Tina L Beckett, et al.
Journal of Alzheimer'S Disease : JAD|July 7, 2012
Postmortem Pittsburgh Compound B (PiB) binding increases with Alzheimer's disease progressionTina L Beckett, Robin L Webb, Dana M Niedowicz, et al.
Neurobiology of Disease|May 25, 2010
Effects of nonsteroidal anti-inflammatory drugs on amyloid-beta pathology in mouse skeletal muscleTina L Beckett, Dana M Niedowicz, Christa M Studzinski, et al.
Acta Neuropathologica Communications|October 8, 2020
Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutationsMeixiang Huang, Erica Modeste, Eric Dammer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 6, 2014
FUS is phosphorylated by DNA-PK and accumulates in the cytoplasm after DNA damageQiudong Deng, Christopher J Holler, Georgia Taylor, et al.
Molecular Neurodegeneration|June 26, 2016
Trehalose upregulates progranulin expression in human and mouse models of GRN haploinsufficiency: a novel therapeutic lead to treat frontotemporal dementiaChristopher J Holler, Georgia Taylor, Zachary T McEachin, et al.
The American Journal of Pathology|November 15, 2011
BACE2 expression increases in human neurodegenerative diseaseChristopher J Holler, Robin L Webb, Ashley L Laux, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Eneuro|August 23, 2017
Intracellular Proteolysis of Progranulin Generates Stable, Lysosomal Granulins that Are Haploinsufficient in Patients with Frontotemporal Dementia Caused by <i>GRN</i> MutationsChristopher J Holler, Georgia Taylor, Qiudong Deng, et al.
Protein Science : a Publication of the Protein Society|June 14, 2017
Disulfide bonds and disorder in granulin-3: An unusual handshake between structural stability and plasticityGaurav Ghag, Christopher J Holler, Georgia Taylor, et al.
Journal of Neurochemistry|December 9, 2009
BACE1 and BACE2 enzymatic activities in Alzheimer's diseaseRachel R Ahmed, Christopher J Holler, Robin L Webb, et al.
Journal of Alzheimer'S Disease : JAD|July 16, 2014
Bridging integrator 1 (BIN1) protein expression increases in the Alzheimer's disease brain and correlates with neurofibrillary tangle pathologyChristopher J Holler, Paulina R Davis, Tina L Beckett, et al.
Journal of Alzheimer'S Disease : JAD|July 7, 2012
Postmortem Pittsburgh Compound B (PiB) binding increases with Alzheimer's disease progressionTina L Beckett, Robin L Webb, Dana M Niedowicz, et al.
Neurobiology of Disease|May 25, 2010
Effects of nonsteroidal anti-inflammatory drugs on amyloid-beta pathology in mouse skeletal muscleTina L Beckett, Dana M Niedowicz, Christa M Studzinski, et al.
Acta Neuropathologica Communications|October 8, 2020
Network analysis of the progranulin-deficient mouse brain proteome reveals pathogenic mechanisms shared in human frontotemporal dementia caused by GRN mutationsMeixiang Huang, Erica Modeste, Eric Dammer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 6, 2014
FUS is phosphorylated by DNA-PK and accumulates in the cytoplasm after DNA damageQiudong Deng, Christopher J Holler, Georgia Taylor, et al.
Molecular Neurodegeneration|June 26, 2016
Trehalose upregulates progranulin expression in human and mouse models of GRN haploinsufficiency: a novel therapeutic lead to treat frontotemporal dementiaChristopher J Holler, Georgia Taylor, Zachary T McEachin, et al.
The American Journal of Pathology|November 15, 2011
BACE2 expression increases in human neurodegenerative diseaseChristopher J Holler, Robin L Webb, Ashley L Laux, et al.
Pageof 2