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ACS Chemical Neuroscience
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February 23, 2019
Increased Secondary Nucleation Underlies Accelerated Aggregation of the Four-Residue N-Terminally Truncated Aβ42 Species Aβ5-42
Tanja Weiffert, Georg Meisl, Patrick Flagmeier, et al.
Biochemistry
|
April 11, 2014
Rare individual amyloid-β oligomers act on astrocytes to initiate neuronal damage
Priyanka Narayan, Kira M Holmström, Dong-Hyun Kim, et al.
Journal of the American Chemical Society
|
October 4, 2021
Accelerating Reaction Rates of Biomolecules by Using Shear Stress in Artificial Capillary Systems
Tuuli A Hakala, Emma V Yates, Pavan K Challa, et al.
Molecules (Basel, Switzerland)
|
February 25, 2022
The Pathological G51D Mutation in Alpha-Synuclein Oligomers Confers Distinct Structural Attributes and Cellular Toxicity
Catherine K Xu, Marta Castellana-Cruz, Serene W Chen, et al.
ACS Nano
|
October 30, 2018
Quantifying Co-Oligomer Formation by α-Synuclein
Marija Iljina, Alexander J Dear, Gonzalo A Garcia, et al.
Nature Chemistry
|
April 15, 2020
Dynamics of oligomer populations formed during the aggregation of Alzheimer's Aβ42 peptide
Thomas C T Michaels, Andela Šarić, Samo Curk, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
July 22, 2015
Force generation by the growth of amyloid aggregates
Therese W Herling, Gonzalo A Garcia, Thomas C T Michaels, et al.
Nano Letters
|
November 2, 2018
Mapping Surface Hydrophobicity of α-Synuclein Oligomers at the Nanoscale
Ji-Eun Lee, Jason C Sang, Margarida Rodrigues, et al.
ACS Chemical Biology
|
November 9, 2013
Single point mutations induce a switch in the molecular mechanism of the aggregation of the Alzheimer's disease associated Aβ42 peptide
Benedetta Bolognesi, Samuel I A Cohen, Pablo Aran Terol, et al.
Nature Chemistry
|
April 19, 2020
Author Correction: Dynamics of oligomer populations formed during the aggregation of Alzheimer's Aβ42 peptide
Thomas C T Michaels, Andela Šarić, Samo Curk, et al.
Page
of 52
Search research articles
Search
Showing results (431-440 of 516) with videos related to
Sort By:
Page
of 52
ACS Chemical Neuroscience
|
February 23, 2019
Increased Secondary Nucleation Underlies Accelerated Aggregation of the Four-Residue N-Terminally Truncated Aβ42 Species Aβ5-42
Tanja Weiffert, Georg Meisl, Patrick Flagmeier, et al.
Biochemistry
|
April 11, 2014
Rare individual amyloid-β oligomers act on astrocytes to initiate neuronal damage
Priyanka Narayan, Kira M Holmström, Dong-Hyun Kim, et al.
Journal of the American Chemical Society
|
October 4, 2021
Accelerating Reaction Rates of Biomolecules by Using Shear Stress in Artificial Capillary Systems
Tuuli A Hakala, Emma V Yates, Pavan K Challa, et al.
Molecules (Basel, Switzerland)
|
February 25, 2022
The Pathological G51D Mutation in Alpha-Synuclein Oligomers Confers Distinct Structural Attributes and Cellular Toxicity
Catherine K Xu, Marta Castellana-Cruz, Serene W Chen, et al.
ACS Nano
|
October 30, 2018
Quantifying Co-Oligomer Formation by α-Synuclein
Marija Iljina, Alexander J Dear, Gonzalo A Garcia, et al.
Nature Chemistry
|
April 15, 2020
Dynamics of oligomer populations formed during the aggregation of Alzheimer's Aβ42 peptide
Thomas C T Michaels, Andela Šarić, Samo Curk, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
July 22, 2015
Force generation by the growth of amyloid aggregates
Therese W Herling, Gonzalo A Garcia, Thomas C T Michaels, et al.
Nano Letters
|
November 2, 2018
Mapping Surface Hydrophobicity of α-Synuclein Oligomers at the Nanoscale
Ji-Eun Lee, Jason C Sang, Margarida Rodrigues, et al.
ACS Chemical Biology
|
November 9, 2013
Single point mutations induce a switch in the molecular mechanism of the aggregation of the Alzheimer's disease associated Aβ42 peptide
Benedetta Bolognesi, Samuel I A Cohen, Pablo Aran Terol, et al.
Nature Chemistry
|
April 19, 2020
Author Correction: Dynamics of oligomer populations formed during the aggregation of Alzheimer's Aβ42 peptide
Thomas C T Michaels, Andela Šarić, Samo Curk, et al.
Page
of 52