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Christopher M Norris

Showing results (11-20 of 66) with videos related to

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Plos One|May 5, 2016
The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell LossL Creed Pettigrew, Richard J Kryscio, Christopher M Norris
Neuroscience Letters|December 23, 2009
Disparate effects of serum on basal and evoked NFAT activity in primary astrocyte culturesJennifer L Furman, Irina A Artiushin, Christopher M Norris
Biochimica Et Biophysica Acta|January 24, 2012
Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's diseaseOlivier Thibault, Tristano Pancani, Philip W Landfield, et al.
Journal of Neurochemistry|December 10, 2017
An emerging role of astrocytes in vascular contributions to cognitive impairment and dementiaBrittani R Price, Christopher M Norris, Pradoldej Sompol, et al.
Neuroreport|May 30, 2024
Elevated calcineurin activity in primary astrocytes leads to the dephosphorylation of connexin 43 in conjunction with increased membrane permeabilityBlaine E Weiss, Susan D Kraner, Irina A Artiushin, et al.
Aging Cell|October 26, 2010
Proteolysis of calcineurin is increased in human hippocampus during mild cognitive impairment and is stimulated by oligomeric Abeta in primary cell cultureHafiz Mohmmad Abdul, Irfan Baig, Harry Levine, et al.
Experimental Neurology|November 27, 2015
Pycnogenol protects CA3-CA1 synaptic function in a rat model of traumatic brain injuryChristopher M Norris, Pradoldej Sompol, Kelly N Roberts, et al.
Molecular and Cellular Pharmacology|April 20, 2010
NFATs and Alzheimer's DiseaseHafiz Mohmmad Abdul, Jennifer L Furman, Michelle A Sama, et al.
Frontiers in Neuroscience|November 22, 2019
Brain-Blood Partition Coefficient and Cerebral Blood Flow in Canines Using Calibrated Short TR Recovery (CaSTRR) Correction MethodScott W Thalman, David K Powell, Margo Ubele, et al.
Scientific Reports|April 26, 2020
Neuroligin-1 is altered in the hippocampus of Alzheimer's disease patients and mouse models, and modulates the toxicity of amyloid-beta oligomersJulien Dufort-Gervais, Chloé Provost, Laurence Charbonneau, et al.
Pageof 7

Showing results (11-20 of 66) with videos related to

Sort By:
Pageof 7
Plos One|May 5, 2016
The TNFα-Transgenic Rat: Hippocampal Synaptic Integrity, Cognition, Function, and Post-Ischemic Cell LossL Creed Pettigrew, Richard J Kryscio, Christopher M Norris
Neuroscience Letters|December 23, 2009
Disparate effects of serum on basal and evoked NFAT activity in primary astrocyte culturesJennifer L Furman, Irina A Artiushin, Christopher M Norris
Biochimica Et Biophysica Acta|January 24, 2012
Reduction in neuronal L-type calcium channel activity in a double knock-in mouse model of Alzheimer's diseaseOlivier Thibault, Tristano Pancani, Philip W Landfield, et al.
Journal of Neurochemistry|December 10, 2017
An emerging role of astrocytes in vascular contributions to cognitive impairment and dementiaBrittani R Price, Christopher M Norris, Pradoldej Sompol, et al.
Neuroreport|May 30, 2024
Elevated calcineurin activity in primary astrocytes leads to the dephosphorylation of connexin 43 in conjunction with increased membrane permeabilityBlaine E Weiss, Susan D Kraner, Irina A Artiushin, et al.
Aging Cell|October 26, 2010
Proteolysis of calcineurin is increased in human hippocampus during mild cognitive impairment and is stimulated by oligomeric Abeta in primary cell cultureHafiz Mohmmad Abdul, Irfan Baig, Harry Levine, et al.
Experimental Neurology|November 27, 2015
Pycnogenol protects CA3-CA1 synaptic function in a rat model of traumatic brain injuryChristopher M Norris, Pradoldej Sompol, Kelly N Roberts, et al.
Molecular and Cellular Pharmacology|April 20, 2010
NFATs and Alzheimer's DiseaseHafiz Mohmmad Abdul, Jennifer L Furman, Michelle A Sama, et al.
Frontiers in Neuroscience|November 22, 2019
Brain-Blood Partition Coefficient and Cerebral Blood Flow in Canines Using Calibrated Short TR Recovery (CaSTRR) Correction MethodScott W Thalman, David K Powell, Margo Ubele, et al.
Scientific Reports|April 26, 2020
Neuroligin-1 is altered in the hippocampus of Alzheimer's disease patients and mouse models, and modulates the toxicity of amyloid-beta oligomersJulien Dufort-Gervais, Chloé Provost, Laurence Charbonneau, et al.
Pageof 7