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Translational Cancer Research|April 16, 2019
Do p53 stress responses impact organismal aging?Paul Hasty, Judith Campisi, Z Dave SharpMethods in Molecular Biology (Clifton, N.J.)|July 20, 2007
Methods to detect biomarkers of cellular senescence: the senescence-associated beta-galactosidase assayKoji Itahana, Judith Campisi, Goberdhan P DimriThe EMBO Journal|March 15, 2011
p38MAPK is a novel DNA damage response-independent regulator of the senescence-associated secretory phenotypeAdam Freund, Christopher K Patil, Judith CampisiBiogerontology|May 13, 2004
Mechanisms of cellular senescence in human and mouse cellsKoji Itahana, Judith Campisi, Goberdhan P DimriExperimental Cell Research|July 10, 2004
Telomere length mediates the effects of telomerase on the cellular response to genotoxic stressMiguel A Rubio, Albert R Davalos, Judith CampisiDevelopmental Cell|January 14, 2015
Of flies, mice, and men: evolutionarily conserved tissue damage responses and agingJoana Neves, Marco Demaria, Judith Campisi, et al.The Journal of Biological Chemistry|May 30, 2002
Reversible manipulation of telomerase expression and telomere length. Implications for the ionizing radiation response and replicative senescence of human cellsMiguel A Rubio, Sahn-Ho Kim, Judith CampisiACS Chemical Biology|February 25, 2022
Engineering Antibodies Targeting p16 MHC-Peptide ComplexesNicholas J Rettko, Judith Campisi, James A WellsOncogene|February 19, 2002
Telomeres, aging and cancer: in search of a happy endingSahn-ho Kim Sh, Patrick Kaminker, Judith CampisiAging Cell|March 23, 2019
Systemic clearance of p16INK4a -positive senescent cells mitigates age-associated intervertebral disc degenerationPrashanti Patil, Qing Dong, Dong Wang, et al.Pageof 25