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Journal of Agricultural and Food Chemistry|March 10, 2012
Isoobtusilactone A sensitizes human hepatoma Hep G2 cells to TRAIL-induced apoptosis via ROS and CHOP-mediated up-regulation of DR5Chung-Yi Chen, Shuenn-Jiun Yiin, Jue-Liang Hsu, et al.Chemical & Pharmaceutical Bulletin|January 8, 2008
Cytotoxic constituents from the leaves of Cinnamomum subaveniumSoong-Yu Kuo, Tian-Jye Hsieh, Yau-Der Wang, et al.International Journal of Molecular Sciences|March 29, 2023
Ginger-Derived 3HDT Exerts Antiproliferative Effects on Breast Cancer Cells by Apoptosis and DNA DamageChung-Yi Chen, Yan-Ning Chen, Jun-Ping Shiau, et al.Planta Medica|December 8, 2025
Threo-2,3-bis-(4-hydroxy-3-methoxyphenyl)-3-methoxypropanol (THMP) Inhibits Bladder Cancer Cell Proliferation via Oxidative Stress-dependent Apoptosis and DNA DamageChe-Wei Yang, Chung-Yi Chen, Chia-Hung Yen, et al.Journal of Natural Products|December 1, 2004
Annoglabayin, a novel dimeric kaurane diterpenoid, and apoptosis in Hep G2 cells of annomontacin from the fruits of Annona glabraChing-Hsein Chen, Tian-Jye Hsieh, Tsan-Zon Liu, et al.European Journal of Pharmacology|August 21, 2007
Isokotomolide A, a new butanolide extracted from the leaves of Cinnamomum kotoense, arrests cell cycle progression and induces apoptosis through the induction of p53/p21 and the initiation of mitochondrial system in human non-small cell lung cancer A549 cellsChung-Yi Chen, Ya-Ling Hsu, Yin-Yi Chen, et al.Molecules (Basel, Switzerland)|April 12, 2012
Bio-functional constituents from the stems of Liriodendron tulipiferaChien-Chih Chiu, Han-Lin Chou, Pei-Fang Wu, et al.Natural Product Research|November 3, 2011
A novel homosesquiterpenoid from the stems of Cinnamomum burmaniiChung-Yi Chen, Zi-Ling Hong, Woei-Ling Yang, et al.Journal of Natural Products|March 11, 2009
Antiallergic potential on RBL-2H3 cells of some phenolic constituents of Zingiber officinale (ginger)Bing-Hung Chen, Pei-Yu Wu, Kuan-Ming Chen, et al.International Journal of Molecular Sciences|December 16, 2014
Pheophytin a inhibits inflammation via suppression of LPS-induced nitric oxide synthase-2, prostaglandin E2, and interleukin-1β of macrophagesChun-Yu Lin, Chien-Hsing Lee, Yu-Wei Chang, et al.Pageof 13