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Chunjiang He

Showing results (41-50 of 54) with videos related to

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Journal of the American College of Cardiology|November 5, 2016
Patient-Specific and Genome-Edited Induced Pluripotent Stem Cell-Derived Cardiomyocytes Elucidate Single-Cell Phenotype of Brugada SyndromePing Liang, Karim Sallam, Haodi Wu, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 8, 2012
MiR-495 is a tumor-suppressor microRNA down-regulated in MLL-rearranged leukemiaXi Jiang, Hao Huang, Zejuan Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 27, 2013
miR-9 is an essential oncogenic microRNA specifically overexpressed in mixed lineage leukemia-rearranged leukemiaPing Chen, Colles Price, Zejuan Li, et al.
Blood|December 25, 2012
PBX3 is an important cofactor of HOXA9 in leukemogenesisZejuan Li, Zhiyu Zhang, Yuanyuan Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 3, 2013
TET1 plays an essential oncogenic role in MLL-rearranged leukemiaHao Huang, Xi Jiang, Zejuan Li, et al.
Cancer Cell|October 20, 2012
Blockade of miR-150 maturation by MLL-fusion/MYC/LIN-28 is required for MLL-associated leukemiaXi Jiang, Hao Huang, Zejuan Li, et al.
Nature Communications|February 23, 2012
miR-196b directly targets both HOXA9/MEIS1 oncogenes and FAS tumour suppressor in MLL-rearranged leukaemiaZejuan Li, Hao Huang, Ping Chen, et al.
Nature Communications|April 11, 2018
Publisher Correction: miR-196b directly targets both HOXA9/MEIS1 oncogenes and FAS tumour suppressor in MLL-rearranged leukaemiaZejuan Li, Hao Huang, Ping Chen, et al.
Nature Communications|February 11, 2018
Author Correction: Targeted inhibition of STAT/TET1 axis as a therapeutic strategy for acute myeloid leukemiaXi Jiang, Chao Hu, Kyle Ferchen, et al.
Blood|January 19, 2012
Up-regulation of a HOXA-PBX3 homeobox-gene signature following down-regulation of miR-181 is associated with adverse prognosis in patients with cytogenetically abnormal AMLZejuan Li, Hao Huang, Yuanyuan Li, et al.
Pageof 6

Showing results (41-50 of 54) with videos related to

Sort By:
Pageof 6
Journal of the American College of Cardiology|November 5, 2016
Patient-Specific and Genome-Edited Induced Pluripotent Stem Cell-Derived Cardiomyocytes Elucidate Single-Cell Phenotype of Brugada SyndromePing Liang, Karim Sallam, Haodi Wu, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 8, 2012
MiR-495 is a tumor-suppressor microRNA down-regulated in MLL-rearranged leukemiaXi Jiang, Hao Huang, Zejuan Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 27, 2013
miR-9 is an essential oncogenic microRNA specifically overexpressed in mixed lineage leukemia-rearranged leukemiaPing Chen, Colles Price, Zejuan Li, et al.
Blood|December 25, 2012
PBX3 is an important cofactor of HOXA9 in leukemogenesisZejuan Li, Zhiyu Zhang, Yuanyuan Li, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 3, 2013
TET1 plays an essential oncogenic role in MLL-rearranged leukemiaHao Huang, Xi Jiang, Zejuan Li, et al.
Cancer Cell|October 20, 2012
Blockade of miR-150 maturation by MLL-fusion/MYC/LIN-28 is required for MLL-associated leukemiaXi Jiang, Hao Huang, Zejuan Li, et al.
Nature Communications|February 23, 2012
miR-196b directly targets both HOXA9/MEIS1 oncogenes and FAS tumour suppressor in MLL-rearranged leukaemiaZejuan Li, Hao Huang, Ping Chen, et al.
Nature Communications|April 11, 2018
Publisher Correction: miR-196b directly targets both HOXA9/MEIS1 oncogenes and FAS tumour suppressor in MLL-rearranged leukaemiaZejuan Li, Hao Huang, Ping Chen, et al.
Nature Communications|February 11, 2018
Author Correction: Targeted inhibition of STAT/TET1 axis as a therapeutic strategy for acute myeloid leukemiaXi Jiang, Chao Hu, Kyle Ferchen, et al.
Blood|January 19, 2012
Up-regulation of a HOXA-PBX3 homeobox-gene signature following down-regulation of miR-181 is associated with adverse prognosis in patients with cytogenetically abnormal AMLZejuan Li, Hao Huang, Yuanyuan Li, et al.
Pageof 6