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Proceedings of the National Academy of Sciences of the United States of America|March 28, 2002
The procaspase-8 isoform, procaspase-8L, recruited to the BAP31 complex at the endoplasmic reticulumDavid G Breckenridge, Mai Nguyen, Stephan Kuppig, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 10, 2004
Bap29/31 influences the intracellular traffic of MHC class I moleculesMarie-Eve Paquet, Myrna Cohen-Doyle, Gordon C Shore, et al.
Molecular and Cellular Biology|March 17, 2004
The yeast split-ubiquitin membrane protein two-hybrid screen identifies BAP31 as a regulator of the turnover of endoplasmic reticulum-associated protein tyrosine phosphatase-like BBing Wang, Jerry Pelletier, Michel J Massaad, et al.
The Journal of Biological Chemistry|July 8, 2011
Glucocorticoid elevation of dexamethasone-induced gene 2 (Dig2/RTP801/REDD1) protein mediates autophagy in lymphocytesJason K Molitoris, Karen S McColl, Sarah Swerdlow, et al.
Oncogene|November 25, 2003
Regulation of apoptosis by endoplasmic reticulum pathwaysDavid G Breckenridge, Marc Germain, Jaigi P Mathai, et al.
Oncotarget|November 5, 2017
Novel NAPRT specific antibody identifies small cell lung cancer and neuronal cancers as promising clinical indications for a NAMPT inhibitor/niacin co-administration strategyJonathan Cole, Marie-Christine Guiot, Michel Gravel, et al.
Molecular Cell|September 20, 2015
MAPL SUMOylation of Drp1 Stabilizes an ER/Mitochondrial Platform Required for Cell DeathJulien Prudent, Rodolfo Zunino, Ayumu Sugiura, et al.
Cancer Research|August 23, 2014
Synergy between the NAMPT inhibitor GMX1777(8) and pemetrexed in non-small cell lung cancer cells is mediated by PARP activation and enhanced NAD consumptionManuel Chan, Michel Gravel, Alexandre Bramoullé, et al.
Tetrahedron Letters|May 29, 2015
Tailored fragments of roseophilin selectively antagonize Mcl-1 <i>in vitro</i>Jack D Bracken, Andrew D Carlson, James H Frederich, et al.
The Journal of Biological Chemistry|January 17, 2003
Uncleaved BAP31 in association with A4 protein at the endoplasmic reticulum is an inhibitor of Fas-initiated release of cytochrome c from mitochondriaBing Wang, Mai Nguyen, David G Breckenridge, et al.
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