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Human Molecular Genetics|February 21, 2012
Bcl-2-associated autophagy regulator Naf-1 required for maintenance of skeletal muscleNatasha C Chang, Mai Nguyen, Johanne Bourdon, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 9, 2014
Translation initiation factor eIF4F modifies the dexamethasone response in multiple myelomaFrancis Robert, William Roman, Alexandre Bramoullé, et al.
The Journal of Cell Biology|July 21, 2004
Bcl-2 functionally interacts with inositol 1,4,5-trisphosphate receptors to regulate calcium release from the ER in response to inositol 1,4,5-trisphosphateRui Chen, Ignacio Valencia, Fei Zhong, et al.
Biochimica Et Biophysica Acta. Molecular Cell Research|January 13, 2022
A non-canonical role for pyruvate kinase M2 as a functional modulator of Ca<sup>2+</sup> signalling through IP<sub>3</sub> receptorsAndrew R Lavik, Karen S McColl, Fernanda O Lemos, et al.
Molecular Cell|July 29, 2008
Targeting Bcl-2-IP3 receptor interaction to reverse Bcl-2's inhibition of apoptotic calcium signalsYi-Ping Rong, Ademuyiwa S Aromolaran, Geert Bultynck, et al.
Cancer Research|May 20, 2016
Novel Protein Disulfide Isomerase Inhibitor with Anticancer Activity in Multiple MyelomaSergei Vatolin, James G Phillips, Babal K Jha, et al.
The Journal of Biological Chemistry|May 27, 2011
Mitochondrion-dependent N-terminal processing of outer membrane Mcl-1 protein removes an essential Mule/Lasu1 protein-binding siteMatthew R Warr, John R Mills, Mai Nguyen, et al.
The EMBO Journal|August 19, 2006
Dissociating the dual roles of apoptosis-inducing factor in maintaining mitochondrial structure and apoptosisEric C C Cheung, Nicholas Joza, Nancy A E Steenaart, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 28, 2007
Small molecule obatoclax (GX15-070) antagonizes MCL-1 and overcomes MCL-1-mediated resistance to apoptosisMai Nguyen, Richard C Marcellus, Anne Roulston, et al.
Nature Cell Biology|September 20, 2011
Bcl-xL regulates metabolic efficiency of neurons through interaction with the mitochondrial F1FO ATP synthaseKambiz N Alavian, Hongmei Li, Leon Collis, et al.
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