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Biochemical and Biophysical Research Communications|December 25, 2012
Acidosis blocks CCAAT/enhancer-binding protein homologous protein (CHOP)- and c-Jun-mediated induction of p53-upregulated mediator of apoptosis (PUMA) during amino acid starvationChristopher B Ryder, Karen McColl, Clark W Distelhorst
The Journal of Biological Chemistry|June 12, 2012
Acidosis promotes Bcl-2 family-mediated evasion of apoptosis: involvement of acid-sensing G protein-coupled receptor Gpr65 signaling to Mek/ErkChristopher Ryder, Karen McColl, Fei Zhong, et al.
Molecular Endocrinology (Baltimore, Md.)|January 18, 2011
Glucocorticoid-mediated repression of the oncogenic microRNA cluster miR-17~92 contributes to the induction of Bim and initiation of apoptosisJason K Molitoris, Karen S McColl, Clark W Distelhorst
Biochimica Et Biophysica Acta|December 6, 2008
Targeting Bcl-2 based on the interaction of its BH4 domain with the inositol 1,4,5-trisphosphate receptorYi-Ping Rong, Paul Barr, Vivien C Yee, et al.
The Journal of Cell Biology|January 5, 2006
Bcl-2 differentially regulates Ca2+ signals according to the strength of T cell receptor activationFei Zhong, Michael C Davis, Karen S McColl, et al.
Autophagy|March 26, 2008
Apoptosis inhibition by Bcl-2 gives way to autophagy in glucocorticoid-treated lymphocytesSarah Swerdlow, Karen McColl, Yiping Rong, et al.
The Journal of Biological Chemistry|April 5, 2003
Microarray analysis uncovers the induction of the proapoptotic BH3-only protein Bim in multiple models of glucocorticoid-induced apoptosisZhengqi Wang, Michael H Malone, Huiling He, et al.
The Journal of Biological Chemistry|December 13, 2002
Bcl-2 on the endoplasmic reticulum regulates Bax activity by binding to BH3-only proteinsMichael J Thomenius, Nancy S Wang, Edmunds Z Reineks, et al.
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