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Annual Review of Cell and Developmental Biology|October 6, 2020
Developing Therapies for Neurodegenerative Disorders: Insights from Protein Aggregation and Cellular Stress ResponsesGiovanna R Mallucci, David Klenerman, David C RubinszteinTraffic (Copenhagen, Denmark)|January 10, 2008
The itinerary of autophagosomes: from peripheral formation to kiss-and-run fusion with lysosomesLuca Jahreiss, Fiona M Menzies, David C RubinszteinThe Journal of Experimental Medicine|June 24, 2015
Therapeutic targeting of autophagy in neurodegenerative and infectious diseasesDavid C Rubinsztein, Carla F Bento, Vojo DereticNature Reviews. Neuroscience|May 21, 2015
Compromised autophagy and neurodegenerative diseasesFiona M Menzies, Angeleen Fleming, David C RubinszteinNature Reviews. Molecular Cell Biology|July 15, 2018
Autophagy as a promoter of longevity: insights from model organismsMalene Hansen, David C Rubinsztein, David W WalkerNature Reviews. Molecular Cell Biology|July 27, 2018
Publisher Correction: Autophagy as a promoter of longevity: insights from model organismsMalene Hansen, David C Rubinsztein, David W WalkerHuman Molecular Genetics|January 8, 2008
Wild-type PABPN1 is anti-apoptotic and reduces toxicity of the oculopharyngeal muscular dystrophy mutationJanet E Davies, Sovan Sarkar, David C RubinszteinSTAR Protocols|May 17, 2021
Transient siRNA-mediated protein knockdown in mouse followed by feeding/starving cycle and liver tissue analysisLidia Wrobel, Farah H Siddiqi, David C RubinszteinAutophagy|August 29, 2022
SMER28 binding to VCP/p97 enhances both autophagic and proteasomal neurotoxic protein clearanceLidia Wrobel, Sandra M Hill, David C RubinszteinThe International Journal of Biochemistry & Cell Biology|March 15, 2006
Oculopharyngeal muscular dystrophy: potential therapies for an aggregate-associated disorderJanet E Davies, Zdenek Berger, David C RubinszteinPageof 32