Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Claudio Hetz

Showing results (161-170 of 201) with videos related to

Pageof 21
Sort By:
Nature Communications|December 17, 2013
A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in miceAníbal I Acuña, Magdalena Esparza, Carlos Kramm, et al.
Archives of Toxicology|April 22, 2014
The transcription factor CHOP, a central component of the transcriptional regulatory network induced upon CCl4 intoxication in mouse liver, is not a critical mediator of hepatotoxicityGisela Campos, Wolfgang Schmidt-Heck, Ahmed Ghallab, et al.
Neurobiology of Disease|August 3, 2025
Expression of amyotrophic lateral sclerosis associated protein disulfide isomerase A3 D217N variant recapitulates early morphological alterations at the neuromuscular junctionMartin Sepulveda, Francisca MartinezTraub, Patricia Ojeda, et al.
Acta Neuropathologica Communications|September 14, 2019
The p75<sup>NTR</sup> neurotrophin receptor is required to organize the mature neuromuscular synapse by regulating synaptic vesicle availabilityViviana Pérez, Francisca Bermedo-Garcia, Diego Zelada, et al.
Journal of Alzheimer'S Disease : JAD|February 22, 2021
DEF8 and Autophagy-Associated Genes Are Altered in Mild Cognitive Impairment, Probable Alzheimer's Disease Patients, and a Transgenic Model of the DiseaseEsteban Leyton, Diego Matus, Sandra Espinoza, et al.
Neurobiology of Disease|May 9, 2025
Expression of protein disulfide isomerase A3Q481K variant associated with amyotrophic lateral sclerosis triggers disease features in miceMartin Sepulveda, Francisca Martínez Traub, Patricia Ojeda, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|January 12, 2025
Artificial enforcement of the unfolded protein response reduces disease features in multiple preclinical models of ALS/FTDVicente Valenzuela, Daniela Becerra, José I Astorga, et al.
Acta Neuropathologica|March 26, 2017
IRE1 signaling exacerbates Alzheimer's disease pathogenesisClaudia Duran-Aniotz, Victor Hugo Cornejo, Sandra Espinoza, et al.
Life Science Alliance|February 23, 2022
Stress-induced tyrosine phosphorylation of RtcB modulates IRE1 activity and signaling outputsAlexandra Papaioannou, Federica Centonze, Alice Metais, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 4, 2017
Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein <i>via</i> the MVB/lysosomal pathwayAlexis E González, Vanessa C Muñoz, Viviana A Cavieres, et al.
Pageof 21

Showing results (161-170 of 201) with videos related to

Sort By:
Pageof 21
Nature Communications|December 17, 2013
A failure in energy metabolism and antioxidant uptake precede symptoms of Huntington's disease in miceAníbal I Acuña, Magdalena Esparza, Carlos Kramm, et al.
Archives of Toxicology|April 22, 2014
The transcription factor CHOP, a central component of the transcriptional regulatory network induced upon CCl4 intoxication in mouse liver, is not a critical mediator of hepatotoxicityGisela Campos, Wolfgang Schmidt-Heck, Ahmed Ghallab, et al.
Neurobiology of Disease|August 3, 2025
Expression of amyotrophic lateral sclerosis associated protein disulfide isomerase A3 D217N variant recapitulates early morphological alterations at the neuromuscular junctionMartin Sepulveda, Francisca MartinezTraub, Patricia Ojeda, et al.
Acta Neuropathologica Communications|September 14, 2019
The p75<sup>NTR</sup> neurotrophin receptor is required to organize the mature neuromuscular synapse by regulating synaptic vesicle availabilityViviana Pérez, Francisca Bermedo-Garcia, Diego Zelada, et al.
Journal of Alzheimer'S Disease : JAD|February 22, 2021
DEF8 and Autophagy-Associated Genes Are Altered in Mild Cognitive Impairment, Probable Alzheimer's Disease Patients, and a Transgenic Model of the DiseaseEsteban Leyton, Diego Matus, Sandra Espinoza, et al.
Neurobiology of Disease|May 9, 2025
Expression of protein disulfide isomerase A3Q481K variant associated with amyotrophic lateral sclerosis triggers disease features in miceMartin Sepulveda, Francisca Martínez Traub, Patricia Ojeda, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|January 12, 2025
Artificial enforcement of the unfolded protein response reduces disease features in multiple preclinical models of ALS/FTDVicente Valenzuela, Daniela Becerra, José I Astorga, et al.
Acta Neuropathologica|March 26, 2017
IRE1 signaling exacerbates Alzheimer's disease pathogenesisClaudia Duran-Aniotz, Victor Hugo Cornejo, Sandra Espinoza, et al.
Life Science Alliance|February 23, 2022
Stress-induced tyrosine phosphorylation of RtcB modulates IRE1 activity and signaling outputsAlexandra Papaioannou, Federica Centonze, Alice Metais, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|March 4, 2017
Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein <i>via</i> the MVB/lysosomal pathwayAlexis E González, Vanessa C Muñoz, Viviana A Cavieres, et al.
Pageof 21