Showing results (11-20 of 34) with videos related to

Sort By:
Pageof 4
Biochimica Et Biophysica Acta|February 19, 2013
TGF-β1 prevents simulated ischemia/reperfusion-induced cardiac fibroblast apoptosis by activation of both canonical and non-canonical signaling pathwaysRaúl Vivar, Claudio Humeres, Pedro Ayala, et al.
Biochemical and Biophysical Research Communications|October 10, 2015
Insulin/NFκB protects against ischemia-induced necrotic cardiomyocyte deathAriel Díaz, Claudio Humeres, Verónica González, et al.
Communications Biology|November 29, 2025
Macrophage ITGAV is dispensable for post-infarction remodeling in mice and does not mediate fibronectin responsesRuoshui Li, Shuaibo Huang, Anis Hanna, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 13, 2022
The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarctionJun Li, Ruoshui Li, Izabela Tuleta, et al.
Biochimica Et Biophysica Acta|November 1, 2015
FoxO1 mediates TGF-beta1-dependent cardiac myofibroblast differentiationRaúl Vivar, Claudio Humeres, Claudia Muñoz, et al.
Journal of Molecular and Cellular Cardiology|May 16, 2019
Distinct roles of myofibroblast-specific Smad2 and Smad3 signaling in repair and remodeling of the infarcted heartShuaibo Huang, Bijun Chen, Ya Su, et al.
The American Journal of Pathology|March 8, 2016
Myocardial Galectin-3 Expression Is Associated with Remodeling of the Pressure-Overloaded Heart and May Delay the Hypertrophic Response without Affecting Survival, Dysfunction, and Cardiac FibrosisOlga Frunza, Ilaria Russo, Amit Saxena, et al.
Frontiers in Pharmacology|December 18, 2018
IFN-β Plays Both Pro- and Anti-inflammatory Roles in the Rat Cardiac Fibroblast Through Differential STAT Protein ActivationSamir Bolívar, Renatto Anfossi, Claudio Humeres, et al.
Cellular Signalling|March 16, 2021
FoxO1 is required for high glucose-dependent cardiac fibroblasts into myofibroblast phenoconversionRaúl Vivar, Renatto Anfossi, Claudio Humeres, et al.
Experimental and Molecular Pathology|April 28, 2012
Cardiac fibroblast death by ischemia/reperfusion is partially inhibited by IGF-1 through both PI3K/Akt and MEK-ERK pathwaysRaúl Vivar, Claudio Humeres, Marcelo Varela, et al.
Pageof 4