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International Journal of Molecular Sciences|February 13, 2026
IL-17 Cytokines Induce IκBζ in Dermal Fibroblasts to Promote Pro-Inflammatory Gene Expression in PsoriasisLejla Svraka, Anna Skarnvad Andersen, Toke Touborg, et al.
Experimental Dermatology|February 23, 2022
Climatotherapy at the Dead Sea for psoriasis is a highly effective anti-inflammatory treatment in the short term: An immunohistochemical studyThomas Emmanuel, Annita Petersen, Hannah Inez Houborg, et al.
Clinical and Experimental Dermatology|April 15, 2024
ERAP1 and ERAP2 gene variants as potential clinical biomarkers of anti-interleukin-17A response in psoriasis vulgarisLasse Kronborg, Emma Oxlund Hansen, Trine Bertelsen, et al.
European Journal of Dermatology : EJD|February 11, 2005
Signal transduction pathways in human epidermisLars Iversen, Claus Johansen, Knud Kragballe
Annals of Dermatology|May 3, 2021
I-Kappa-B-Zeta Regulates Interleukin-17A/Tumor Necrosis Factor-Alpha Mediated Synergistic Induction of Interleukin-19 and Interleukin-20 in Humane KeratinocytesTrine Bertelsen, Lars Iversen, Claus Johansen
Experimental Dermatology|June 26, 2018
The human IL-17A/F heterodimer regulates psoriasis-associated genes through IκBζTrine Bertelsen, Lars Iversen, Claus Johansen
Cytokine|December 15, 2024
When recombinant proteins go wrong: The hidden pitfall of recombinant protein contaminationLejla Svraka, Hakim Ben Abdallah, Claus Johansen
The Journal of Investigative Dermatology|February 20, 2025
The Differential Roles of HSP90 Isoforms in Skin Inflammation: Anti-Inflammatory Potential of TRAP1 InhibitionHakim Ben Abdallah, Lars Iversen, Claus Johansen
Psoriasis (Auckland, N.Z.)|July 8, 2021
Key Signaling Pathways in Psoriasis: Recent Insights from Antipsoriatic TherapeuticsHakim Ben Abdallah, Claus Johansen, Lars Iversen
The Journal of Investigative Dermatology|May 22, 2025
Oral Roflumilast Suppresses Proinflammatory Cytokine Signaling and Reduces CD4+ T-Cell and Neutrophil Infiltration in PsoriasisElena Baez, Mette Gyldenløve, Hakim Ben Abdallah, et al.
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