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The Journal of Clinical Investigation|July 15, 2021
ALKBH1 reduces DNA N6-methyladenine to allow for vascular calcification in chronic kidney diseaseKe Zhu, Jochen ReiserPflugers Archiv : European Journal of Physiology|July 10, 2017
suPAR and chronic kidney disease-a podocyte storyMartin Zeier, Jochen ReiserAnnual Review of Medicine|November 30, 2012
Podocyte biology and pathogenesis of kidney diseaseJochen Reiser, Sanja SeverKidney International|November 20, 2009
Proteinuria: an enzymatic disease of the podocyte?Peter Mundel, Jochen ReiserCurrent Hypertension Reports|January 8, 2008
Dual effects of RAS blockade on blood pressure and podocyte functionJochen Reiser, Peter MundelNature Reviews. Nephrology|December 31, 2013
Proteinuria: abate or applaud abatacept in proteinuric kidney disease?Jochen Reiser, Nada AlachkarPediatric Nephrology (Berlin, Germany)|December 17, 2010
TRPC channel modulation in podocytes-inching toward novel treatments for glomerular diseaseShafic El Hindi, Jochen ReiserThe Journal of Pathology|December 15, 2018
More expression, less function: cleaved dynamin in glomerular kidney diseaseMehmet M Altintas, Jochen ReiserAmerican Journal of Physiology. Renal Physiology|August 6, 2010
TRPC6 channels and their binding partners in podocytes: role in glomerular filtration and pathophysiologyStuart E Dryer, Jochen ReiserPageof 21