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Plos One|April 4, 2014
CD8α dendritic cells drive establishment of HSV-1 latencyKevin R Mott, Sariah J Allen, Mandana Zandian, et al.Cornea|November 12, 2015
Confocal Microscopic Analysis of a Rabbit Eye Model of High-Incidence Recurrent Herpes Stromal KeratitisJames V Jester, Naoyuki Morishige, Lbachir BenMohamed, et al.Journal of Virology|July 26, 2019
The Vaccinia Virus (VACV) B1 and Cellular VRK2 Kinases Promote VACV Replication Factory Formation through Phosphorylation-Dependent Inhibition of VACV B12Amber B Rico, Zhigang Wang, Annabel T Olson, et al.The Journal of General Virology|September 12, 2006
Herpes simplex virus type 1 ICP0 localizes in the stromal layer of infected rabbit corneas and resides predominantly in the cytoplasm and/or perinuclear region of rabbit keratocytesNaoyuki Morishige, James V Jester, Julie Naito, et al.Journal of Neurovirology|April 25, 2007
Reactivation phenotype in rabbits of a herpes simplex virus type 1 mutant containing an unrelated antiapoptosis gene in place of latency-associated transcriptLing Jin, Guey-Chuen Perng, Dale Carpenter, et al.Journal of Virology|January 12, 2022
Progesterone Sporadically Induces Reactivation from Latency in Female Calves but Proficiently Stimulates Bovine Herpesvirus 1 Productive InfectionFouad S El-Mayet, Gabriela Toomer, Jeffery B Ostler, et al.Journal of the American Veterinary Medical Association|September 2, 2009
Efficacy of a combination viral vaccine for protection of cattle against experimental infection with field isolates of bovine herpesvirus-1John A Ellis, Sheryl P Gow, Noriko Goji, et al.Journal of Neurovirology|April 6, 2006
A mutant deleted for most of the herpes simplex virus type 1 (HSV-1) UOL gene does not affect the spontaneous reactivation phenotype in rabbitsDavid Chan, Jacob Cohen, Julie Naito, et al.Blood|September 4, 2025
Innate lymphoid cells prevent graft-versus-host disease via IL-9-driven T-cell senescenceDejene Milkessa Tufa, Eric Hoffmeyer, Kristin L Schaller, et al.The Journal of General Virology|October 24, 2003
The bovine herpesvirus-1 LR ORF2 is critical for this gene's ability to restore the high wild-type reactivation phenotype to a herpes simplex virus-1 LAT null mutantKevin R Mott, Nelson Osorio, Ling Jin, et al.Pageof 16