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Journal of Experimental Neuroscience|December 15, 2017
Breaking the Cycle, Cholesterol Cycling, and Synapse Damage in Response to Amyloid-βClive BateBiochimica Et Biophysica Acta. Molecular Basis of Disease|December 17, 2017
Cholesterol ester hydrolase inhibitors reduce the production of synaptotoxic amyloid-β oligomersHarriet McHale-Owen, Clive BateJournal of Cell Science|March 29, 2018
The phospholipase A2 pathway controls a synaptic cholesterol ester cycle and synapse damageCraig Osborne, Ewan West, Clive BateNeuropharmacology|June 28, 2015
An in vitro model for synaptic loss in neurodegenerative diseases suggests a neuroprotective role for valproic acid via inhibition of cPLA2 dependent signallingRobin S B Williams, Clive BateJournal of Cell Science|August 2, 2017
The cholesterol ester cycle regulates signalling complexes and synapse damage caused by amyloid-βEwan West, Craig Osborne, Clive BateNeuropharmacology|September 27, 2017
Valproic acid and its congener propylisopropylacetic acid reduced the amount of soluble amyloid-β oligomers released from 7PA2 cellsRobin S B Williams, Clive BateThe Biochemical Journal|July 22, 2017
Sialylated glycosylphosphatidylinositols suppress the production of toxic amyloid-β oligomersWilliam Nolan, Harriet McHale-Owen, Clive BateBiology|June 5, 2015
Monoacylated Cellular Prion Proteins Reduce Amyloid-β-Induced Activation of Cytoplasmic Phospholipase A2 and Synapse DamageEwan West, Craig Osborne, William Nolan, et al.Biochemistry|October 3, 2012
The N-methylated peptide SEN304 powerfully inhibits Aβ(1-42) toxicity by perturbing oligomer formationHozefa Amijee, Clive Bate, Alun Williams, et al.The Journal of Laryngology and Otology|May 1, 2004
Early experience with the Mills sleeve prosthesis for reconstruction of the incus long processRobert Mills, Alun WilliamsPageof 15