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Current Opinion in Hematology|March 15, 2019
A somatic missense mutation in GNAQ causes capillary malformationColette Bichsel, Joyce BischoffCells|February 15, 2022
Generation of an hiPSC-Derived Co-Culture System to Assess the Effects of Neuroinflammation on Blood-Brain Barrier IntegrityDaniel Bull, Christophe Schweitzer, Colette Bichsel, et al.Angiogenesis|September 29, 2024
Similarities and differences between brain and skin GNAQ p.R183Q driven capillary malformationsSana Nasim, Colette Bichsel, Anna Pinto, et al.Biorxiv : the Preprint Server for Biology|July 1, 2024
Similarities and differences between brain and skin <i>GNAQ</i> p.R183Q driven capillary malformationsSana Nasim, Colette Bichsel, Anna Pinto, et al.Journal of Personalized Medicine|February 25, 2022
Microphysiological Neurovascular Barriers to Model the Inner Retinal MicrovasculatureThomas L Maurissen, Georgios Pavlou, Colette Bichsel, et al.Acta Neuropathologica Communications|March 27, 2024
MRC1 and LYVE1 expressing macrophages in vascular beds of GNAQ p.R183Q driven capillary malformations in Sturge Weber syndromeSana Nasim, Colette Bichsel, Stephen Dayneka, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|October 21, 2021
Endothelial <i>GNAQ</i> p.R183Q Increases ANGPT2 (Angiopoietin-2) and Drives Formation of Enlarged Blood VesselsLan Huang, Colette Bichsel, Alexis L Norris, et al.Pageof 1