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Current Opinion in Hematology|March 15, 2019
A somatic missense mutation in GNAQ causes capillary malformationColette Bichsel, Joyce BischoffAngiogenesis|September 29, 2024
Similarities and differences between brain and skin GNAQ p.R183Q driven capillary malformationsSana Nasim, Colette Bichsel, Anna Pinto, et al.Biorxiv : the Preprint Server for Biology|July 1, 2024
Similarities and differences between brain and skin GNAQ p.R183Q driven capillary malformationsSana Nasim, Colette Bichsel, Anna Pinto, et al.Acta Neuropathologica Communications|March 27, 2024
MRC1 and LYVE1 expressing macrophages in vascular beds of GNAQ p.R183Q driven capillary malformations in Sturge Weber syndromeSana Nasim, Colette Bichsel, Stephen Dayneka, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|October 21, 2021
Endothelial GNAQ p.R183Q Increases ANGPT2 (Angiopoietin-2) and Drives Formation of Enlarged Blood VesselsLan Huang, Colette Bichsel, Alexis L Norris, et al.Trends in Cardiovascular Medicine|August 6, 2002
Monoclonal expansion of endothelial cells in hemangioma: an intrinsic defect with extrinsic consequences?Joyce BischoffThe Journal of Craniofacial Surgery|January 27, 2009
Progenitor cells in infantile hemangiomaJoyce BischoffCells|February 15, 2022
Generation of an hiPSC-Derived Co-Culture System to Assess the Effects of Neuroinflammation on Blood-Brain Barrier IntegrityDaniel Bull, Christophe Schweitzer, Colette Bichsel, et al.Bio-Protocol|March 3, 2021
Isolation of Stem Cells, Endothelial Cells and Pericytes from Human Infantile HemangiomaLan Huang, Joyce BischoffArteriosclerosis, Thrombosis, and Vascular Biology|July 1, 2021
Endothelial-Mesenchymal Transition in Cardiovascular DiseaseZahra Alvandi, Joyce BischoffPageof 13