Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Colin G Nichols

Showing results (91-100 of 219) with videos related to

Pageof 22
Sort By:
The Journal of Physiology|August 20, 2005
A difference in inward rectification and polyamine block and permeation between the Kir2.1 and Kir3.1/Kir3.4 K+ channelsSamy M Y Makary, Tom W Claydon, Decha Enkvetchakul, et al.
Diabetes|October 27, 2006
Expression of ATP-insensitive KATP channels in pancreatic beta-cells underlies a spectrum of diabetic phenotypesJoseph C Koster, Maria S Remedi, Ricard Masia, et al.
The Journal of Biological Chemistry|September 28, 2004
Functional characterization of a prokaryotic Kir channelDecha Enkvetchakul, Jaya Bhattacharyya, Iana Jeliazkova, et al.
The Journal of Biological Chemistry|January 10, 2013
Scanning the topography of polyamine blocker binding in an inwardly rectifying potassium channelHarley T Kurata, Alejandro Akrouh, Jenny B W Li, et al.
Journal of Molecular and Cellular Cardiology|September 12, 2009
Differential K(ATP) channel pharmacology in intact mouse heartAlexey V Glukhov, Thomas P Flagg, Vadim V Fedorov, et al.
Pediatric Diabetes|August 7, 2009
Successful sulfonylurea treatment of an insulin-naïve neonate with diabetes mellitus due to a KCNJ11 mutationJennifer A Wambach, Bess A Marshall, Joseph C Koster, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 16, 2011
Mechanism for selectivity-inactivation coupling in KcsA potassium channelsWayland W L Cheng, Jason G McCoy, Ameer N Thompson, et al.
Diabetes|January 30, 2007
An ATP-binding mutation (G334D) in KCNJ11 is associated with a sulfonylurea-insensitive form of developmental delay, epilepsy, and neonatal diabetesRicard Masia, Joseph C Koster, Stefano Tumini, et al.
Physiological Genomics|December 3, 2009
Genes controlling postural changes in blood pressure: comprehensive association analysis of ATP-sensitive potassium channel genes KCNJ8 and ABCC9Justine A Ellis, Angela Lamantia, Raul Chavez, et al.
Channels (Austin, Tex.)|May 8, 2012
Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunitsYu-Wen Lin, Alejandro Akrouh, YeouChing Hsu, et al.
Pageof 22

Showing results (91-100 of 219) with videos related to

Sort By:
Pageof 22
The Journal of Physiology|August 20, 2005
A difference in inward rectification and polyamine block and permeation between the Kir2.1 and Kir3.1/Kir3.4 K+ channelsSamy M Y Makary, Tom W Claydon, Decha Enkvetchakul, et al.
Diabetes|October 27, 2006
Expression of ATP-insensitive KATP channels in pancreatic beta-cells underlies a spectrum of diabetic phenotypesJoseph C Koster, Maria S Remedi, Ricard Masia, et al.
The Journal of Biological Chemistry|September 28, 2004
Functional characterization of a prokaryotic Kir channelDecha Enkvetchakul, Jaya Bhattacharyya, Iana Jeliazkova, et al.
The Journal of Biological Chemistry|January 10, 2013
Scanning the topography of polyamine blocker binding in an inwardly rectifying potassium channelHarley T Kurata, Alejandro Akrouh, Jenny B W Li, et al.
Journal of Molecular and Cellular Cardiology|September 12, 2009
Differential K(ATP) channel pharmacology in intact mouse heartAlexey V Glukhov, Thomas P Flagg, Vadim V Fedorov, et al.
Pediatric Diabetes|August 7, 2009
Successful sulfonylurea treatment of an insulin-naïve neonate with diabetes mellitus due to a KCNJ11 mutationJennifer A Wambach, Bess A Marshall, Joseph C Koster, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 16, 2011
Mechanism for selectivity-inactivation coupling in KcsA potassium channelsWayland W L Cheng, Jason G McCoy, Ameer N Thompson, et al.
Diabetes|January 30, 2007
An ATP-binding mutation (G334D) in KCNJ11 is associated with a sulfonylurea-insensitive form of developmental delay, epilepsy, and neonatal diabetesRicard Masia, Joseph C Koster, Stefano Tumini, et al.
Physiological Genomics|December 3, 2009
Genes controlling postural changes in blood pressure: comprehensive association analysis of ATP-sensitive potassium channel genes KCNJ8 and ABCC9Justine A Ellis, Angela Lamantia, Raul Chavez, et al.
Channels (Austin, Tex.)|May 8, 2012
Compound heterozygous mutations in the SUR1 (ABCC 8) subunit of pancreatic K(ATP) channels cause neonatal diabetes by perturbing the coupling between Kir6.2 and SUR1 subunitsYu-Wen Lin, Alejandro Akrouh, YeouChing Hsu, et al.
Pageof 22