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Plos One
|
December 19, 2013
Fibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathy
Sarah N Cilvik, Joy I Wang, Kory J Lavine, et al.
The Journal of Biological Chemistry
|
November 22, 2013
Control of KirBac3.1 potassium channel gating at the interface between cytoplasmic domains
Lejla Zubcevic, Vassiliy N Bavro, Joao R C Muniz, et al.
Journal of Cell Science
|
February 27, 2014
Tuning the electrical properties of the heart by differential trafficking of KATP ion channel complexes
Eric C Arakel, Sören Brandenburg, Keita Uchida, et al.
Circulation Research
|
November 22, 2008
Ca2+-independent alterations in diastolic sarcomere length and relaxation kinetics in a mouse model of lipotoxic diabetic cardiomyopathy
Thomas P Flagg, Olivier Cazorla, Maria S Remedi, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
June 2, 2016
K(ATP) channel gain-of-function leads to increased myocardial L-type Ca(2+) current and contractility in Cantu syndrome
Mark D Levin, Gautam K Singh, Hai Xia Zhang, et al.
Clinical Endocrinology
|
January 4, 2014
Focal congenital hyperinsulinism managed by medical treatment: a diagnostic algorithm based on molecular genetic screening
Arianna Maiorana, Fabrizio Barbetti, Arianna Boiani, et al.
Diabetes
|
October 29, 2010
Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 (KCNJ11) mutation V290M mutation: K(ATP) channel inactivation mechanism and clinical management
Karen J Loechner, Alejandro Akrouh, Harley T Kurata, et al.
The Journal of Physiology
|
May 7, 2020
Kir6.1-dependent K<sub>ATP</sub> channels in lymphatic smooth muscle and vessel dysfunction in mice with Kir6.1 gain-of-function
Michael J Davis, Hae Jin Kim, Scott D Zawieja, et al.
The Journal of Biological Chemistry
|
September 30, 2010
Functional complementation and genetic deletion studies of KirBac channels: activatory mutations highlight gating-sensitive domains
Jennifer J Paynter, Isabelle Andres-Enguix, Philip W Fowler, et al.
Hormone Research in Paediatrics
|
January 9, 2022
Sulfonylurea-Insensitive Permanent Neonatal Diabetes Caused by a Severe Gain-of-Function Tyr330His Substitution in Kir6.2
Conor McClenaghan, Novella Rapini, Domenico Umberto De Rose, et al.
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of 22
Search research articles
Search
Showing results (191-200 of 219) with videos related to
Sort By:
Page
of 22
Plos One
|
December 19, 2013
Fibroblast growth factor receptor 1 signaling in adult cardiomyocytes increases contractility and results in a hypertrophic cardiomyopathy
Sarah N Cilvik, Joy I Wang, Kory J Lavine, et al.
The Journal of Biological Chemistry
|
November 22, 2013
Control of KirBac3.1 potassium channel gating at the interface between cytoplasmic domains
Lejla Zubcevic, Vassiliy N Bavro, Joao R C Muniz, et al.
Journal of Cell Science
|
February 27, 2014
Tuning the electrical properties of the heart by differential trafficking of KATP ion channel complexes
Eric C Arakel, Sören Brandenburg, Keita Uchida, et al.
Circulation Research
|
November 22, 2008
Ca2+-independent alterations in diastolic sarcomere length and relaxation kinetics in a mouse model of lipotoxic diabetic cardiomyopathy
Thomas P Flagg, Olivier Cazorla, Maria S Remedi, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
June 2, 2016
K(ATP) channel gain-of-function leads to increased myocardial L-type Ca(2+) current and contractility in Cantu syndrome
Mark D Levin, Gautam K Singh, Hai Xia Zhang, et al.
Clinical Endocrinology
|
January 4, 2014
Focal congenital hyperinsulinism managed by medical treatment: a diagnostic algorithm based on molecular genetic screening
Arianna Maiorana, Fabrizio Barbetti, Arianna Boiani, et al.
Diabetes
|
October 29, 2010
Congenital hyperinsulinism and glucose hypersensitivity in homozygous and heterozygous carriers of Kir6.2 (KCNJ11) mutation V290M mutation: K(ATP) channel inactivation mechanism and clinical management
Karen J Loechner, Alejandro Akrouh, Harley T Kurata, et al.
The Journal of Physiology
|
May 7, 2020
Kir6.1-dependent K<sub>ATP</sub> channels in lymphatic smooth muscle and vessel dysfunction in mice with Kir6.1 gain-of-function
Michael J Davis, Hae Jin Kim, Scott D Zawieja, et al.
The Journal of Biological Chemistry
|
September 30, 2010
Functional complementation and genetic deletion studies of KirBac channels: activatory mutations highlight gating-sensitive domains
Jennifer J Paynter, Isabelle Andres-Enguix, Philip W Fowler, et al.
Hormone Research in Paediatrics
|
January 9, 2022
Sulfonylurea-Insensitive Permanent Neonatal Diabetes Caused by a Severe Gain-of-Function Tyr330His Substitution in Kir6.2
Conor McClenaghan, Novella Rapini, Domenico Umberto De Rose, et al.
Page
of 22