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Colin H Peters

Showing results (11-20 of 26) with videos related to

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The Journal of General Physiology|June 23, 2020
E1784K, the most common Brugada syndrome and long-QT syndrome type 3 mutant, disrupts sodium channel inactivation through two separate mechanismsColin H Peters, Abeline R Watkins, Olivia L Poirier, et al.
Plos One|September 13, 2017
Depolarization of the conductance-voltage relationship in the NaV1.5 mutant, E1784K, is due to altered fast inactivationColin H Peters, Alec Yu, Wandi Zhu, et al.
The Journal of Biological Chemistry|January 4, 2013
Proton sensors in the pore domain of the cardiac voltage-gated sodium channelDavid K Jones, Colin H Peters, Charlene R Allard, et al.
Journal of Neurophysiology|September 19, 2019
Case studies in neuroscience: a novel amino acid duplication in the NH<sub>2</sub>-terminus of the brain sodium channel Na<sub>V</sub>1.1 underlying Dravet syndromeMadeline Angus, Colin H Peters, Damon Poburko, et al.
Frontiers in Cellular Neuroscience|January 11, 2020
Functional Genomics of Epilepsy and Associated Neurodevelopmental Disorders Using Simple Animal Models: From Genes, Molecules to Brain NetworksRichard Rosch, Dominic R W Burrows, Laura B Jones, et al.
The Journal of General Physiology|October 28, 2022
The funny current If is essential for the fight-or-flight response in cardiac pacemaker cellsColin H Peters, Christian Rickert, Stefano Morotti, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 14, 2021
Bidirectional flow of the funny current (I<sub>f</sub>) during the pacemaking cycle in murine sinoatrial node myocytesColin H Peters, Pin W Liu, Stefano Morotti, et al.
Frontiers in Pharmacology|December 20, 2021
The L1624Q Variant in <i>SCN1A</i> Causes Familial Epilepsy Through a Mixed Gain and Loss of Channel FunctionLaura B Jones, Colin H Peters, Richard E Rosch, et al.
International Journal of Molecular Sciences|June 2, 2021
Intracellular Na<sup>+</sup> Modulates Pacemaking Activity in Murine Sinoatrial Node Myocytes: An <i>In Silico</i> AnalysisStefano Morotti, Haibo Ni, Colin H Peters, et al.
Brain Communications|November 10, 2021
Persistent sodium currents in <i>SCN1A</i> developmental and degenerative epileptic dyskinetic encephalopathyKathleen M Gorman, Colin H Peters, Bryan Lynch, et al.
Pageof 3

Showing results (11-20 of 26) with videos related to

Sort By:
Pageof 3
The Journal of General Physiology|June 23, 2020
E1784K, the most common Brugada syndrome and long-QT syndrome type 3 mutant, disrupts sodium channel inactivation through two separate mechanismsColin H Peters, Abeline R Watkins, Olivia L Poirier, et al.
Plos One|September 13, 2017
Depolarization of the conductance-voltage relationship in the NaV1.5 mutant, E1784K, is due to altered fast inactivationColin H Peters, Alec Yu, Wandi Zhu, et al.
The Journal of Biological Chemistry|January 4, 2013
Proton sensors in the pore domain of the cardiac voltage-gated sodium channelDavid K Jones, Colin H Peters, Charlene R Allard, et al.
Journal of Neurophysiology|September 19, 2019
Case studies in neuroscience: a novel amino acid duplication in the NH<sub>2</sub>-terminus of the brain sodium channel Na<sub>V</sub>1.1 underlying Dravet syndromeMadeline Angus, Colin H Peters, Damon Poburko, et al.
Frontiers in Cellular Neuroscience|January 11, 2020
Functional Genomics of Epilepsy and Associated Neurodevelopmental Disorders Using Simple Animal Models: From Genes, Molecules to Brain NetworksRichard Rosch, Dominic R W Burrows, Laura B Jones, et al.
The Journal of General Physiology|October 28, 2022
The funny current If is essential for the fight-or-flight response in cardiac pacemaker cellsColin H Peters, Christian Rickert, Stefano Morotti, et al.
Proceedings of the National Academy of Sciences of the United States of America|July 14, 2021
Bidirectional flow of the funny current (I<sub>f</sub>) during the pacemaking cycle in murine sinoatrial node myocytesColin H Peters, Pin W Liu, Stefano Morotti, et al.
Frontiers in Pharmacology|December 20, 2021
The L1624Q Variant in <i>SCN1A</i> Causes Familial Epilepsy Through a Mixed Gain and Loss of Channel FunctionLaura B Jones, Colin H Peters, Richard E Rosch, et al.
International Journal of Molecular Sciences|June 2, 2021
Intracellular Na<sup>+</sup> Modulates Pacemaking Activity in Murine Sinoatrial Node Myocytes: An <i>In Silico</i> AnalysisStefano Morotti, Haibo Ni, Colin H Peters, et al.
Brain Communications|November 10, 2021
Persistent sodium currents in <i>SCN1A</i> developmental and degenerative epileptic dyskinetic encephalopathyKathleen M Gorman, Colin H Peters, Bryan Lynch, et al.
Pageof 3