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Coral K Wille

Showing results (1-10 of 10) with videos related to

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Stem Cell Reports|January 7, 2022
DOT1L inhibition enhances pluripotency beyond acquisition of epithelial identity and without immediate suppression of the somatic transcriptomeCoral K Wille, Rupa Sridharan
Frontiers in Cell and Developmental Biology|June 23, 2022
Connecting the DOTs on Cell IdentityCoral K Wille, Rupa Sridharan
Stem Cell Reports|November 23, 2023
DOT1L interaction partner AF10 controls patterning of H3K79 methylation and RNA polymerase II to maintain cell identityCoral K Wille, Edwin N Neumann, Aniruddha J Deshpande, et al.
Journal of Virology|December 23, 2016
Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus LatencyCoral K Wille, Yangguang Li, Lixin Rui, et al.
Science Advances|November 17, 2023
DOT1L is a barrier to histone acetylation during reprogramming to pluripotencyCoral K Wille, Xiaoya Zhang, Spencer A Haws, et al.
Journal of Virology|June 24, 2011
Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latencyAmanda R Robinson, Swee Sen Kwek, Stacy R Hagemeier, et al.
Journal of Virology|November 9, 2012
Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replicationCoral K Wille, Dhananjay M Nawandar, Amanda R Panfil, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 15, 2015
5-hydroxymethylation of the EBV genome regulates the latent to lytic switchCoral K Wille, Dhananjay M Nawandar, Amanda N Henning, et al.
Plos Pathogens|October 3, 2015
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial CellsDhananjay M Nawandar, Anqi Wang, Kathleen Makielski, et al.
Molecular Cell|March 26, 2020
Methyl-Metabolite Depletion Elicits Adaptive Responses to Support Heterochromatin Stability and Epigenetic PersistenceSpencer A Haws, Deyang Yu, Cunqi Ye, et al.
Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
Stem Cell Reports|January 7, 2022
DOT1L inhibition enhances pluripotency beyond acquisition of epithelial identity and without immediate suppression of the somatic transcriptomeCoral K Wille, Rupa Sridharan
Frontiers in Cell and Developmental Biology|June 23, 2022
Connecting the DOTs on Cell IdentityCoral K Wille, Rupa Sridharan
Stem Cell Reports|November 23, 2023
DOT1L interaction partner AF10 controls patterning of H3K79 methylation and RNA polymerase II to maintain cell identityCoral K Wille, Edwin N Neumann, Aniruddha J Deshpande, et al.
Journal of Virology|December 23, 2016
Restricted TET2 Expression in Germinal Center Type B Cells Promotes Stringent Epstein-Barr Virus LatencyCoral K Wille, Yangguang Li, Lixin Rui, et al.
Science Advances|November 17, 2023
DOT1L is a barrier to histone acetylation during reprogramming to pluripotencyCoral K Wille, Xiaoya Zhang, Spencer A Haws, et al.
Journal of Virology|June 24, 2011
Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latencyAmanda R Robinson, Swee Sen Kwek, Stacy R Hagemeier, et al.
Journal of Virology|November 9, 2012
Viral genome methylation differentially affects the ability of BZLF1 versus BRLF1 to activate Epstein-Barr virus lytic gene expression and viral replicationCoral K Wille, Dhananjay M Nawandar, Amanda R Panfil, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 15, 2015
5-hydroxymethylation of the EBV genome regulates the latent to lytic switchCoral K Wille, Dhananjay M Nawandar, Amanda N Henning, et al.
Plos Pathogens|October 3, 2015
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial CellsDhananjay M Nawandar, Anqi Wang, Kathleen Makielski, et al.
Molecular Cell|March 26, 2020
Methyl-Metabolite Depletion Elicits Adaptive Responses to Support Heterochromatin Stability and Epigenetic PersistenceSpencer A Haws, Deyang Yu, Cunqi Ye, et al.
Pageof 1