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Proceedings of the National Academy of Sciences of the United States of America|September 13, 2018
Deconvolution of pro- and antiviral genomic responses in Zika virus-infected and bystander macrophagesAaron F Carlin, Edward A Vizcarra, Emilie Branche, et al.
Inflammatory Bowel Diseases|May 11, 2011
SAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitisTheresa T Pizarro, Luca Pastorelli, Giorgos Bamias, et al.
Cardiovascular Research|September 4, 2024
Olfr2-positive macrophages originate from monocytes proliferate in situ and present a pro-inflammatory foamy-like phenotypeSujit Silas Armstrong Suthahar, Felix Sebastian Nettersheim, Ahmad Alimadadi, et al.
Cell Reports|January 4, 2019
High-Affinity Bent β2-Integrin Molecules in Arresting Neutrophils Face Each Other through Binding to ICAMs In cisZhichao Fan, William Bill Kiosses, Hao Sun, et al.
Circulation Research|December 24, 2011
NR4A1 (Nur77) deletion polarizes macrophages toward an inflammatory phenotype and increases atherosclerosisRichard N Hanna, Iftach Shaked, Harper G Hubbeling, et al.
Blood|August 16, 2017
CD177 modulates human neutrophil migration through activation-mediated integrin and chemoreceptor regulationMing Bai, Ricardo Grieshaber-Bouyer, Junxia Wang, et al.
European Journal of Cell Biology|March 14, 2022
HS1 deficiency protects against sepsis by attenuating neutrophil-inflicted lung damageIdaira M Guerrero-Fonseca, Alexander García-Ponce, Eduardo Vadillo, et al.
International Journal of Molecular Sciences|February 13, 2025
CITE-Seq Analysis Reveals a Differential Natural Killer Cell SPON2 Expression in Cardiovascular Disease Patients Impacted by Human-Cytomegalovirus Serostatus and DiabetesSujit Silas Armstrong, Daniel G Chen, Sunil Kumar, et al.
The Journal of Clinical Investigation|August 14, 2012
Dynamic T cell-APC interactions sustain chronic inflammation in atherosclerosisEkaterina K Koltsova, Zacarias Garcia, Grzegorz Chodaczek, et al.
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