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Cristiane Cecatto

Showing results (1-10 of 25) with videos related to

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Journal of Neurochemistry|January 24, 2016
2-Methylcitric acid impairs glutamate metabolism and induces permeability transition in brain mitochondriaAlexandre Umpierrez Amaral, Cristiane Cecatto, Roger Frigério Castilho, et al.
Neurotoxicity Research|February 22, 2014
Ethylmalonic acid induces permeability transition in isolated brain mitochondriaCristiane Cecatto, Alexandre Umpierrez Amaral, Guilhian Leipnitz, et al.
Toxicology in Vitro : an International Journal Published in Association with BIBRA|October 20, 2019
Impairment of mitochondrial bioenergetics and permeability transition induction caused by major long-chain fatty acids accumulating in VLCAD deficiency in skeletal muscle as potential pathomechanisms of myopathyCristiane Cecatto, Alexandre Umpierrez Amaral, Ana Cristina Roginski, et al.
The FEBS Journal|September 27, 2015
Deregulation of mitochondrial functions provoked by long-chain fatty acid accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial permeability transition deficiencies in rat heart--mitochondrial permeability transition pore opening as a potential contributing pathomechanism of cardiac alterations in these disordersCristiane Cecatto, Fernanda H Hickmann, Marília D N Rodrigues, et al.
Journal of Cellular Biochemistry|June 21, 2018
High vulnerability of the heart and liver to 3-hydroxypalmitic acid-induced disruption of mitochondrial functions in intact cell systemsCristiane Cecatto, Alessandro Wajner, Carmen Regla Vargas, et al.
Mitochondrion|October 27, 2019
Disturbance of mitochondrial functions associated with permeability transition pore opening induced by cis-5-tetradecenoic and myristic acids in liver of adolescent ratsCristiane Cecatto, Alexandre Umpierrez Amaral, Alessandro Wajner, et al.
Toxicology in Vitro : an International Journal Published in Association with BIBRA|July 3, 2016
Disturbance of mitochondrial functions provoked by the major long-chain 3-hydroxylated fatty acids accumulating in MTP and LCHAD deficiencies in skeletal muscleCristiane Cecatto, Kálita Dos Santos Godoy, Janaína Camacho da Silva, et al.
Molecular Neurobiology|November 5, 2017
S-Adenosylmethionine Promotes Oxidative Stress and Decreases Na<sup>+</sup>, K<sup>+</sup>-ATPase Activity in Cerebral Cortex Supernatants of Adolescent Rats: Implications for the Pathogenesis of S-Adenosylhomocysteine Hydrolase DeficiencyÂngela Zanatta, Cristiane Cecatto, Rafael Teixeira Ribeiro, et al.
Biochimica Et Biophysica Acta|April 15, 2015
Uncoupling, metabolic inhibition and induction of mitochondrial permeability transition in rat liver mitochondria caused by the major long-chain hydroxyl monocarboxylic fatty acids accumulating in LCHAD deficiencyFernanda Hermes Hickmann, Cristiane Cecatto, Daniele Kleemann, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|January 14, 2020
Disturbance of bioenergetics and calcium homeostasis provoked by metabolites accumulating in propionic acidemia in heart mitochondria of developing ratsAna Cristina Roginski, Alessandro Wajner, Cristiane Cecatto, et al.
Pageof 3

Showing results (1-10 of 25) with videos related to

Sort By:
Pageof 3
Journal of Neurochemistry|January 24, 2016
2-Methylcitric acid impairs glutamate metabolism and induces permeability transition in brain mitochondriaAlexandre Umpierrez Amaral, Cristiane Cecatto, Roger Frigério Castilho, et al.
Neurotoxicity Research|February 22, 2014
Ethylmalonic acid induces permeability transition in isolated brain mitochondriaCristiane Cecatto, Alexandre Umpierrez Amaral, Guilhian Leipnitz, et al.
Toxicology in Vitro : an International Journal Published in Association with BIBRA|October 20, 2019
Impairment of mitochondrial bioenergetics and permeability transition induction caused by major long-chain fatty acids accumulating in VLCAD deficiency in skeletal muscle as potential pathomechanisms of myopathyCristiane Cecatto, Alexandre Umpierrez Amaral, Ana Cristina Roginski, et al.
The FEBS Journal|September 27, 2015
Deregulation of mitochondrial functions provoked by long-chain fatty acid accumulating in long-chain 3-hydroxyacyl-CoA dehydrogenase and mitochondrial permeability transition deficiencies in rat heart--mitochondrial permeability transition pore opening as a potential contributing pathomechanism of cardiac alterations in these disordersCristiane Cecatto, Fernanda H Hickmann, Marília D N Rodrigues, et al.
Journal of Cellular Biochemistry|June 21, 2018
High vulnerability of the heart and liver to 3-hydroxypalmitic acid-induced disruption of mitochondrial functions in intact cell systemsCristiane Cecatto, Alessandro Wajner, Carmen Regla Vargas, et al.
Mitochondrion|October 27, 2019
Disturbance of mitochondrial functions associated with permeability transition pore opening induced by cis-5-tetradecenoic and myristic acids in liver of adolescent ratsCristiane Cecatto, Alexandre Umpierrez Amaral, Alessandro Wajner, et al.
Toxicology in Vitro : an International Journal Published in Association with BIBRA|July 3, 2016
Disturbance of mitochondrial functions provoked by the major long-chain 3-hydroxylated fatty acids accumulating in MTP and LCHAD deficiencies in skeletal muscleCristiane Cecatto, Kálita Dos Santos Godoy, Janaína Camacho da Silva, et al.
Molecular Neurobiology|November 5, 2017
S-Adenosylmethionine Promotes Oxidative Stress and Decreases Na<sup>+</sup>, K<sup>+</sup>-ATPase Activity in Cerebral Cortex Supernatants of Adolescent Rats: Implications for the Pathogenesis of S-Adenosylhomocysteine Hydrolase DeficiencyÂngela Zanatta, Cristiane Cecatto, Rafael Teixeira Ribeiro, et al.
Biochimica Et Biophysica Acta|April 15, 2015
Uncoupling, metabolic inhibition and induction of mitochondrial permeability transition in rat liver mitochondria caused by the major long-chain hydroxyl monocarboxylic fatty acids accumulating in LCHAD deficiencyFernanda Hermes Hickmann, Cristiane Cecatto, Daniele Kleemann, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|January 14, 2020
Disturbance of bioenergetics and calcium homeostasis provoked by metabolites accumulating in propionic acidemia in heart mitochondria of developing ratsAna Cristina Roginski, Alessandro Wajner, Cristiane Cecatto, et al.
Pageof 3