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Frontiers in Physiology|June 21, 2021
PK11195 Protects From Cell Death Only When Applied During Reperfusion: Succinate-Mediated Mechanism of ActionLea K Seidlmayer, Benjamin J Hanson, Phung N Thai, et al.Proceedings of the National Academy of Sciences of the United States of America|April 2, 2015
Novel Epac fluorescent ligand reveals distinct Epac1 vs. Epac2 distribution and function in cardiomyocytesLaëtitia Pereira, Holger Rehmann, Dieu Hung Lao, et al.Biomaterials|May 19, 2012
Controlling the contractile strength of engineered cardiac muscle by hierarchal tissue architectureAdam W Feinberg, Patrick W Alford, Hongwei Jin, et al.American Journal of Physiology. Heart and Circulatory Physiology|December 7, 2010
Phorbol ester and endothelin-1 alter functional expression of Na+/Ca2+ exchange, K+, and Ca2+ currents in cultured neonatal rat myocytesJosé L Puglisi, Weilong Yuan, Valeriy Timofeyev, et al.American Journal of Physiology. Heart and Circulatory Physiology|March 23, 2010
Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failureAndreas Rinne, Nidhi Kapur, Jeffery D Molkentin, et al.Circulation Research|November 13, 2013
Overexpression of the Na+/K+ ATPase α2 but not α1 isoform attenuates pathological cardiac hypertrophy and remodelingRobert N Correll, Petra Eder, Adam R Burr, et al.Nature Communications|June 15, 2024
Kinetics and mapping of Ca-driven calmodulin conformations on skeletal and cardiac muscle ryanodine receptorsRobyn T Rebbeck, Bengt Svensson, Jingyan Zhang, et al.Journal of Molecular and Cellular Cardiology|October 26, 2010
SR-targeted CaMKII inhibition improves SR Ca²+ handling, but accelerates cardiac remodeling in mice overexpressing CaMKIIδCSabine Huke, Jaime Desantiago, Marcia A Kaetzel, et al.Circulation Research|April 5, 2003
The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failureTong Zhang, Lars S Maier, Nancy D Dalton, et al.Journal of Proteome Research|March 28, 2015
CaMKII Phosphorylation of Na(V)1.5: Novel in Vitro Sites Identified by Mass Spectrometry and Reduced S516 Phosphorylation in Human Heart FailureAnthony W Herren, Darren M Weber, Robert R Rigor, et al.Pageof 41