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Annals of the New York Academy of Sciences|November 30, 2006
Regulation of Ca2+ and Na+ in normal and failing cardiac myocytesDonald M Bers, Sanda Despa, Julie BossuytJournal of Molecular and Cellular Cardiology|January 12, 2019
CaMKII signaling in heart diseases: Emerging role in diabetic cardiomyopathyBence Hegyi, Donald M Bers, Julie BossuytBiophysical Journal|June 23, 2016
Stretch-Activated Current Can Promote or Suppress Cardiac Alternans Depending on Voltage-Calcium InteractionSamuel Galice, Donald M Bers, Daisuke SatoCirculation Research|June 7, 2003
Ca2+ scraps: local depletions of free [Ca2+] in cardiac sarcoplasmic reticulum during contractions leave substantial Ca2+ reserveThomas R Shannon, Tao Guo, Donald M BersBiophysical Journal|September 20, 2005
Regulation of cardiac sarcoplasmic reticulum Ca release by luminal [Ca] and altered gating assessed with a mathematical modelThomas R Shannon, Fei Wang, Donald M BersAmerican Journal of Physiology. Heart and Circulatory Physiology|June 18, 2013
Post-translational modifications of the cardiac Na channel: contribution of CaMKII-dependent phosphorylation to acquired arrhythmiasAnthony W Herren, Donald M Bers, Eleonora GrandiAnnals of Biomedical Engineering|July 26, 2008
Quantitative panoramic imaging of epicardial electrical activityQing Lou, Crystal M Ripplinger, Philip V Bayly, et al.Basic Research in Cardiology|December 14, 2002
Upregulated Na/Ca exchange is involved in both contractile dysfunction and arrhythmogenesis in heart failureDonald M Bers, Steven M Pogwizd, Klaus SchlotthauerJournal of Molecular and Cellular Cardiology|September 18, 2002
Frequency-dependent acceleration of relaxation in the heart depends on CaMKII, but not phospholambanJaime DeSantiago, Lars S Maier, Donald M BersProgress in Biophysics and Molecular Biology|May 15, 2004
Modeling the isolated cardiac myocyteJose L Puglisi, Fei Wang, Donald M BersPageof 41