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Molecular and Cellular Endocrinology|March 12, 2019
Maternal exposure to imazalil disrupts the endocrine system in F1 generation miceCuiyuan Jin, Rui Zhang, Zhengwei Fu, et al.
Endocrine Journal|May 13, 2021
Maternal exposure of mice to sodium p-perfluorous nonenoxybenzene sulfonate causes endocrine disruption in both dams and offspringCaiyun Wang, Cuiyuan Jin, Wenqing Tu, et al.
Chemosphere|July 10, 2016
Oral imazalil exposure induces gut microbiota dysbiosis and colonic inflammation in miceCuiyuan Jin, Zhaoyang Zeng, Zhengwei Fu, et al.
Environmental Toxicology|February 17, 2018
Chronic exposure of mice to low doses of imazalil induces hepatotoxicity at the physiological, biochemical, and transcriptomic levelsCuiyuan Jin, Ting Luo, Zhengwei Fu, et al.
Environmental Toxicology|January 3, 2023
Effects of maternal exposure to procymidone on hepatic metabolism in the offspring of miceXiaofang Wang, Lingyu Hu, Cuiyuan Jin, et al.
Environmental Pollution (Barking, Essex : 1987)|November 16, 2017
Exposure to the fungicide propamocarb causes gut microbiota dysbiosis and metabolic disorder in miceSisheng Wu, Cuiyuan Jin, Yueyi Wang, et al.
Journal of Hazardous Materials|August 20, 2020
Maternal exposure to imazalil disrupts intestinal barrier and bile acids enterohepatic circulation tightly related IL-22 expression in F0, F1 and F2 generations of miceCuiyuan Jin, Xianling Yuan, Caiyun Wang, et al.
The Science of the Total Environment|July 29, 2021
Maternal exposure to sodium ρ-perfluorous nonenoxybenzene sulfonate during pregnancy and lactation disrupts intestinal barrier and may cause obstacles to the nutrient transport and metabolism in F0 and F1 generations of miceCaiyun Wang, You Weng, Wenqing Tu, et al.
Pesticide Biochemistry and Physiology|March 7, 2022
Catechin from green tea had the potential to decrease the chlorpyrifos induced oxidative stress in larval zebrafish (Danio rerio)Yun Zhao, Chanlin Fang, Cuiyuan Jin, et al.
Environmental Pollution (Barking, Essex : 1987)|May 22, 2021
6:2 Cl-PFESA has the potential to cause liver damage and induce lipid metabolism disorders in female mice through the action of PPAR-γZihong Pan, Wenyu Miao, Caiyun Wang, et al.
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