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Frontiers in Cellular Neuroscience|March 8, 2021
Emerging Evidence Highlighting the Importance of Redox Dysregulation in the Pathogenesis of Amyotrophic Lateral Sclerosis (ALS)Cyril Jones Jagaraj, Sonam Parakh, Julie D AtkinNeuromolecular Medicine|August 18, 2025
A Slower-Progressing TDP-43 rNLS8 Mouse Model for ALS: Implications for Preclinical and Mechanistic StudiesCyril Jones Jagaraj, Prachi Mehta, Julie Hunter, et al.Cellular and Molecular Life Sciences : CMLS|March 2, 2024
Molecular hallmarks of ageing in amyotrophic lateral sclerosisCyril Jones Jagaraj, Sina Shadfar, Sara Assar Kashani, et al.Brain, Behavior, and Immunity|June 16, 2025
IL-6 trans-signalling is elevated in ALS models and drives TDP-43 induced inflammatory responses in microgliaGrace Risby-Jones, Jianina Marallag, Cyril Jones Jagaraj, et al.Aging Cell|May 15, 2025
The Redox Activity of Protein Disulphide Isomerase Functions in Non-Homologous End-Joining Repair to Prevent DNA DamageSina Shadfar, Fabiha Farzana, Sayanthooran Saravanabavan, et al.Molecular Neurobiology|May 9, 2024
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTDJessica Sultana, Audrey M G Ragagnin, Sonam Parakh, et al.Brain : a Journal of Neurology|March 10, 2026
Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosisCyril Jones Jagaraj, Sayanthooran Saravanabavan, Sonam Parakh, et al.Pageof 1