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D A Compston

Showing results (71-80 of 118) with videos related to

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Neurology|July 27, 2001
Patterns of disease in concordant parent-child pairs with multiple sclerosisR Hupperts, S Broadley, A Mander, et al.
Multiple Sclerosis (Houndmills, Basingstoke, England)|April 25, 2000
Multiple sclerosis and tonsillectomy: no evidence for an influence on the development of disease or clinical phenotypeS A Broadley, J Deans, S J Chataway, et al.
Journal of Neurology|May 1, 1995
Sequence of the human homologue of a mitochondrially encoded murine transplantation antigen in patients with multiple sclerosisR M Chalmers, N Robertson, H Kellar-Wood, et al.
Immunology|May 1, 1992
Oligodendrocytes lack glycolipid anchored proteins which protect them against complement lysis. Restoration of resistance to lysis by incorporation of CD59M G Wing, J Zajicek, D J Seilly, et al.
Annals of Neurology|July 1, 1994
Leber's hereditary optic neuropathy mitochondrial DNA mutations in multiple sclerosisH Kellar-Wood, N Robertson, G G Govan, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|August 1, 1992
EDMUS, a European database for multiple sclerosisC Confavreux, D A Compston, O R Hommes, et al.
Immunology|August 1, 1989
Reversible injury of cultured rat oligodendrocytes by complementN J Scolding, W A Houston, B P Morgan, et al.
Lancet (London, England)|December 5, 1981
Optic neuritis and multiple sclerosis: do factor B alleles influence progression of disease?A H Fielder, J R Batchelor, B N Vakarelis, et al.
Brain : a Journal of Neurology|August 1, 1989
The role of complement in the pathogenesis of experimental allergic encephalomyelitisC Linington, B P Morgan, N J Scolding, et al.
Journal of Neuroimmunology|May 1, 1989
Myelin-oligodendrocyte glycoprotein (MOG) is a surface marker of oligodendrocyte maturationN J Scolding, S Frith, C Linington, et al.
Pageof 12

Showing results (71-80 of 118) with videos related to

Sort By:
Pageof 12
Neurology|July 27, 2001
Patterns of disease in concordant parent-child pairs with multiple sclerosisR Hupperts, S Broadley, A Mander, et al.
Multiple Sclerosis (Houndmills, Basingstoke, England)|April 25, 2000
Multiple sclerosis and tonsillectomy: no evidence for an influence on the development of disease or clinical phenotypeS A Broadley, J Deans, S J Chataway, et al.
Journal of Neurology|May 1, 1995
Sequence of the human homologue of a mitochondrially encoded murine transplantation antigen in patients with multiple sclerosisR M Chalmers, N Robertson, H Kellar-Wood, et al.
Immunology|May 1, 1992
Oligodendrocytes lack glycolipid anchored proteins which protect them against complement lysis. Restoration of resistance to lysis by incorporation of CD59M G Wing, J Zajicek, D J Seilly, et al.
Annals of Neurology|July 1, 1994
Leber's hereditary optic neuropathy mitochondrial DNA mutations in multiple sclerosisH Kellar-Wood, N Robertson, G G Govan, et al.
Journal of Neurology, Neurosurgery, and Psychiatry|August 1, 1992
EDMUS, a European database for multiple sclerosisC Confavreux, D A Compston, O R Hommes, et al.
Immunology|August 1, 1989
Reversible injury of cultured rat oligodendrocytes by complementN J Scolding, W A Houston, B P Morgan, et al.
Lancet (London, England)|December 5, 1981
Optic neuritis and multiple sclerosis: do factor B alleles influence progression of disease?A H Fielder, J R Batchelor, B N Vakarelis, et al.
Brain : a Journal of Neurology|August 1, 1989
The role of complement in the pathogenesis of experimental allergic encephalomyelitisC Linington, B P Morgan, N J Scolding, et al.
Journal of Neuroimmunology|May 1, 1989
Myelin-oligodendrocyte glycoprotein (MOG) is a surface marker of oligodendrocyte maturationN J Scolding, S Frith, C Linington, et al.
Pageof 12