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Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|October 1, 1995
Mitogenesis by v-Src: fluctuations throughout G1 of classical immediate early AP-1 and mitogen-activated protein kinase responses that parallel the need for the oncoproteinA W Wyke, M C Frame, D A Gillespie, et al.Journal of Virology|September 1, 1993
Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activityK L Hawker, A Pintzas, R F Hennigan, et al.Oncogene|August 1, 1994
Transcriptional activation by the v-Jun oncoprotein is independent of positive regulatory phosphorylationE J Black, A D Catling, J R Woodgett, et al.Development (Cambridge, England)|December 1, 1995
Differential regulation of AP-1 and novel TRE-specific DNA-binding complexes during differentiation of oligodendrocyte-type-2-astrocyte (O-2A) progenitor cellsS C Barnett, M Rosario, A Doyle, et al.The New England Journal of Medicine|April 28, 1988
High incidence of poor sulfoxidation in patients with primary biliary cirrhosisA B Olomu, C R Vickers, R H Waring, et al.Gut|April 1, 1991
Role of endoscopic retrograde cholangiopancreatography after orthotopic liver transplantationH J O'Connor, C R Vickers, J A Buckels, et al.Oncogene|July 1, 1993
Gene-regulatory properties of Myc helix-loop-helix/leucine zipper mutants: Max-dependent DNA binding and transcriptional activation in yeast correlates with transforming capacityD H Crouch, F Fisher, W Clark, et al.The EMBO Journal|December 15, 1993
Transcription activation by Myc and Max: flanking sequences target activation to a subset of CACGTG motifs in vivoF Fisher, D H Crouch, P S Jayaraman, et al.Oncogene|July 19, 2011
Cdk-mediated phosphorylation of Chk1 is required for efficient activation and full checkpoint proficiency in response to DNA damageN Xu, S Libertini, E J Black, et al.Molecular and Cellular Biology|March 14, 2000
v-Jun overrides the mitogen dependence of S-phase entry by deregulating retinoblastoma protein phosphorylation and E2F-pocket protein interactions as a consequence of enhanced cyclin E-cdk2 catalytic activityW Clark, E J Black, A MacLaren, et al.Pageof 5