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Neurology
|
July 27, 2005
Familial Alzheimer disease: decreases in CSF Abeta42 levels precede cognitive decline
M Moonis, J M Swearer, M P E Dayaw, et al.
Science (New York, N.Y.)
|
April 21, 1972
Does the striate cortex begin reconstruction of the visual world?
J E Mittenthal, W B Kristan, W G Tatton, et al.
Dementia (Basel, Switzerland)
|
July 1, 1995
Familial Alzheimer's disease and cortical Lewy bodies: is there a genetic susceptibility factor?
C F Lippa, T W Smith, L Nee, et al.
Journal of the American Geriatrics Society
|
July 14, 2000
Postmenopausal hormone therapy and cognitive function in healthy older women
F Grodstein, J Chen, D A Pollen, et al.
Annals of Neurology
|
September 8, 2000
Familial Alzheimer's disease: site of mutation influences clinical phenotype
C F Lippa, J M Swearer, K J Kane, et al.
The American Journal of Pathology
|
November 12, 1998
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes
C F Lippa, H Fujiwara, D M Mann, et al.
Neurology
|
February 1, 1996
Familial and sporadic Alzheimer's disease: neuropathology cannot exclude a final common pathway
C F Lippa, A M Saunders, T W Smith, et al.
Neurobiology of Aging
|
May 30, 2001
Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque deposition
K Ishii, C Lippa, T Tomiyama, et al.
Brain Research. Molecular Brain Research
|
May 29, 1998
Amyloid-beta-protein isoforms in brain of subjects with PS1-linked, beta APP-linked and sporadic Alzheimer disease
A Tamaoka, P E Fraser, K Ishii, et al.
Page
of 4
Search research articles
Search
Showing results (31-40 of 39) with videos related to
Sort By:
Page
of 4
You have reached the last page of results.
This site can display upto 39 results.
Neurology
|
July 27, 2005
Familial Alzheimer disease: decreases in CSF Abeta42 levels precede cognitive decline
M Moonis, J M Swearer, M P E Dayaw, et al.
Science (New York, N.Y.)
|
April 21, 1972
Does the striate cortex begin reconstruction of the visual world?
J E Mittenthal, W B Kristan, W G Tatton, et al.
Dementia (Basel, Switzerland)
|
July 1, 1995
Familial Alzheimer's disease and cortical Lewy bodies: is there a genetic susceptibility factor?
C F Lippa, T W Smith, L Nee, et al.
Journal of the American Geriatrics Society
|
July 14, 2000
Postmenopausal hormone therapy and cognitive function in healthy older women
F Grodstein, J Chen, D A Pollen, et al.
Annals of Neurology
|
September 8, 2000
Familial Alzheimer's disease: site of mutation influences clinical phenotype
C F Lippa, J M Swearer, K J Kane, et al.
The American Journal of Pathology
|
November 12, 1998
Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes
C F Lippa, H Fujiwara, D M Mann, et al.
Neurology
|
February 1, 1996
Familial and sporadic Alzheimer's disease: neuropathology cannot exclude a final common pathway
C F Lippa, A M Saunders, T W Smith, et al.
Neurobiology of Aging
|
May 30, 2001
Distinguishable effects of presenilin-1 and APP717 mutations on amyloid plaque deposition
K Ishii, C Lippa, T Tomiyama, et al.
Brain Research. Molecular Brain Research
|
May 29, 1998
Amyloid-beta-protein isoforms in brain of subjects with PS1-linked, beta APP-linked and sporadic Alzheimer disease
A Tamaoka, P E Fraser, K Ishii, et al.
Page
of 4