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Pharmaceutics
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January 21, 2023
What Can RNA-Based Therapy Do for Monogenic Diseases?
Luka A Clarke, Margarida D Amaral
The Lancet. Respiratory Medicine
|
July 6, 2016
Classification of CFTR mutation classes - Authors' reply
Kris De Boeck, Margarida D Amaral
The FEBS Journal
|
May 18, 2013
Post-translational modifications of CFTR: insight into protein trafficking and cystic fibrosis disease
Margarida D Amaral, Carlos M Farinha
Journal of Neurophysiology
|
August 19, 2007
BDNF induces calcium elevations associated with IBDNF, a nonselective cationic current mediated by TRPC channels
Michelle D Amaral, Lucas Pozzo-Miller
Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society
|
November 1, 2022
Development of novel therapeutics for all individuals with CF (the future goes on)
Margarida D Amaral, Patrick T Harrison
Cellscience
|
January 15, 2010
The dynamics of excitatory synapse formation on dendritic spines
Michelle D Amaral, Lucas Pozzo-Miller
Molecular and Cellular Biology
|
June 1, 2005
Most F508del-CFTR is targeted to degradation at an early folding checkpoint and independently of calnexin
Carlos M Farinha, Margarida D Amaral
General and Comparative Endocrinology
|
January 1, 1983
Effects of insulin and glucagon on plasma glucose levels and glycogen content in organs of the freshwater teleost Pimelodus maculatus
N M Carneiro, A D Amaral
Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society
|
July 22, 2019
Theranostics by testing CFTR modulators in patient-derived materials: The current status and a proposal for subjects with rare CFTR mutations
Margarida D Amaral, Kris de Boeck,
Neural Plasticity
|
July 20, 2012
Intracellular Ca2+ stores and Ca2+ influx are both required for BDNF to rapidly increase quantal vesicular transmitter release
Michelle D Amaral, Lucas Pozzo-Miller
Page
of 31
Search research articles
Search
Showing results (21-30 of 307) with videos related to
Sort By:
Page
of 31
Pharmaceutics
|
January 21, 2023
What Can RNA-Based Therapy Do for Monogenic Diseases?
Luka A Clarke, Margarida D Amaral
The Lancet. Respiratory Medicine
|
July 6, 2016
Classification of CFTR mutation classes - Authors' reply
Kris De Boeck, Margarida D Amaral
The FEBS Journal
|
May 18, 2013
Post-translational modifications of CFTR: insight into protein trafficking and cystic fibrosis disease
Margarida D Amaral, Carlos M Farinha
Journal of Neurophysiology
|
August 19, 2007
BDNF induces calcium elevations associated with IBDNF, a nonselective cationic current mediated by TRPC channels
Michelle D Amaral, Lucas Pozzo-Miller
Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society
|
November 1, 2022
Development of novel therapeutics for all individuals with CF (the future goes on)
Margarida D Amaral, Patrick T Harrison
Cellscience
|
January 15, 2010
The dynamics of excitatory synapse formation on dendritic spines
Michelle D Amaral, Lucas Pozzo-Miller
Molecular and Cellular Biology
|
June 1, 2005
Most F508del-CFTR is targeted to degradation at an early folding checkpoint and independently of calnexin
Carlos M Farinha, Margarida D Amaral
General and Comparative Endocrinology
|
January 1, 1983
Effects of insulin and glucagon on plasma glucose levels and glycogen content in organs of the freshwater teleost Pimelodus maculatus
N M Carneiro, A D Amaral
Journal of Cystic Fibrosis : Official Journal of the European Cystic Fibrosis Society
|
July 22, 2019
Theranostics by testing CFTR modulators in patient-derived materials: The current status and a proposal for subjects with rare CFTR mutations
Margarida D Amaral, Kris de Boeck,
Neural Plasticity
|
July 20, 2012
Intracellular Ca2+ stores and Ca2+ influx are both required for BDNF to rapidly increase quantal vesicular transmitter release
Michelle D Amaral, Lucas Pozzo-Miller
Page
of 31