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Plos Pathogens|December 14, 2012
Clostridium difficile Toxin B causes epithelial cell necrosis through an autoprocessing-independent mechanismNicole M Chumbler, Melissa A Farrow, Lynne A Lapierre, et al.
Anaerobe|January 27, 2022
Persistent bacteremia and psoas abscess caused by a lethal toxin-deficient PaeniclostridiumsordelliiCara D Varley, Lisa M Rogers, Beverly Rea Dixon, et al.
The Journal of Biological Chemistry|November 19, 2005
Structural determinants for the binding of anthrax lethal factor to oligomeric protective antigenRoman A Melnyk, Krissi M Hewitt, D Borden Lacy, et al.
Elife|September 3, 2020
Structural analysis of the Legionella pneumophila Dot/Icm type IV secretion system core complexClarissa L Durie, Michael J Sheedlo, Jeong Min Chung, et al.
Infection and Immunity|April 30, 2008
Helicobacter pylori VacA subdomain required for intracellular toxin activity and assembly of functional oligomeric complexesSusan E Ivie, Mark S McClain, Victor J Torres, et al.
Science Advances|July 19, 2023
Nonsteroidal anti-inflammatory drugs sensitize epithelial cells to Clostridioides difficile toxin-mediated mitochondrial damageJoshua Soto Ocaña, Nile U Bayard, Jessica L Hart, et al.
Biochemistry|June 10, 2010
Reconstitution of Helicobacter pylori VacA toxin from purified componentsChristian González-Rivera, Kelly A Gangwer, Mark S McClain, et al.
ACS Chemical Biology|January 8, 2020
Small Molecule Inhibitor Screen Reveals Calcium Channel Signaling as a Mechanistic Mediator of Clostridium difficile TcdB-Induced NecrosisMelissa A Farrow, Nicole M Chumber, Sarah C Bloch, et al.
Journal of Pediatric Gastroenterology and Nutrition|September 14, 2020
Two-step Testing for Clostridioides Difficile is Inadequate in Differentiating Infection From Colonization in ChildrenJacob M Parnell, Irtiqa Fazili, Sarah C Bloch, et al.
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