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Water Science and Technology : a Journal of the International Association on Water Pollution Research|June 6, 2007
Estimation of kinetic parameters of a model for deammonification in biofilms and evaluation of the modelD Brockmann, K H Rosenwinke, E MorgenrothBiotechnology and Bioengineering|May 31, 2008
Practical identifiability of biokinetic parameters of a model describing two-step nitrification in biofilmsD Brockmann, K-H Rosenwinkel, E MorgenrothGene|October 17, 2001
The histone acetyltransferase activity of PCAF cooperates with the brahma/SWI2-related protein BRG-1 in the activation of the enhancer A of the MHC class I promoterD Brockmann, O Lehmkühler, U Schmücker, et al.The Journal of Biological Chemistry|March 18, 2000
cAMP-independent activation of the adenovirus type 12 E2 promoter correlates with the recruitment of CREB-1/ATF-1, E1A(12S), and CBP to the E2-CREP Fax, K S Lipinski, H Esche, et al.Oncogene|April 18, 1996
Repression of c-Jun-induced mouse major histocompatibility class I promoter (H-2Kb) activity by the Adenovirus type 12-unique 52R E1A proteinD Brockmann, D Schäfer, H C Kirch, et al.The Journal of Biological Chemistry|September 28, 2000
E1A12S-mediated activation of the adenovirus type 12 E2 promoter depends on the histone acetyltransferase activity of p300/CBPP Fax, O Lehmkuhler, C Kuhn, et al.Virology|February 1, 1994
Adenovirus type 12 early region 1A expresses a 52R protein repressing the trans-activating activity of transcription factor c-Jun/AP-1D Brockmann, L Feng, G Kröner, et al.The Journal of General Virology|February 1, 1997
The E1A N terminus (aa 1-29) of the highly oncogenic adenovirus type 12 harbours a trans-activation function not detectable in the non-oncogenic serotype 2K S Lipinski, G Kröner-Lux, H Esche, et al.Gene|July 16, 1990
Cloning of adenovirus type 12 E1 genes into a retroviral vector and their differential splicing in mouse cellsD Brockmann, A Schmidtmann, S Fürst, et al.Oncogene|May 29, 1999
Expression of human p53 requires synergistic activation of transcription from the p53 promoter by AP-1, NF-kappaB and Myc/MaxH C Kirch, S Flaswinkel, H Rumpf, et al.Pageof 4