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D Bruton

Showing results (71-80 of 112) with videos related to

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Plant Disease|March 3, 2019
Effect of Insect Exclusion on the Incidence of Yellow Vine Disease and of the Associated Bacterium in SquashBlake Bextine, Astri Wayadande, B D Bruton, et al.
Acta Physiologica (Oxford, England)|June 15, 2007
Activation of Ca(2+)-dependent protein kinase II during repeated contractions in single muscle fibres from mouse is dependent on the frequency of sarcoplasmic reticulum Ca(2+) releaseJ Aydin, T Korhonen, P Tavi, et al.
Plant Disease|March 3, 2019
Detection of Cucurbit Yellow Vine Disease in Squash and Pumpkin in MassachusettsR L Wick, J Lerner, S D Pair, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 16, 2023
Increased tetanic calcium in early fatigue of mammalian muscle fibers is accompanied by accelerated force development despite a decreased forceCecilia Leijding, Ida Viken, Joseph D Bruton, et al.
Journal of Cellular Physiology|January 30, 2010
Myogenic skeletal muscle satellite cells communicate by tunnelling nanotubesPasi Tavi, Topi Korhonen, Sandra L Hänninen, et al.
Phytopathology|October 24, 2008
Polymerase chain reaction detection and phylogenetic characterization of an agent associated with yellow vine disease of cucurbitsF J Avila, B D Bruton, J Fletcher, et al.
Phytopathology|October 24, 2008
Identification, Phylogenetic Analysis, and Biological Characterization of Serratia marcescens Strains Causing Cucurbit Yellow Vine DiseaseJ Rascoe, M Berg, U Melcher, et al.
Diabetes|July 28, 2005
Insulin and inositol 1,4,5-trisphosphate trigger abnormal cytosolic Ca2+ transients and reveal mitochondrial Ca2+ handling defects in cardiomyocytes of ob/ob miceJérémy Fauconnier, Johanna T Lanner, Shi-Jin Zhang, et al.
Cardiovascular Research|December 13, 2006
Insulin potentiates TRPC3-mediated cation currents in normal but not in insulin-resistant mouse cardiomyocytesJérémy Fauconnier, Johanna T Lanner, Ariane Sultan, et al.
The Journal of General Physiology|January 13, 2019
SR Ca<sup>2+</sup> leak in skeletal muscle fibers acts as an intracellular signal to increase fatigue resistanceNiklas Ivarsson, C Mikael Mattsson, Arthur J Cheng, et al.
Pageof 12

Showing results (71-80 of 112) with videos related to

Sort By:
Pageof 12
Plant Disease|March 3, 2019
Effect of Insect Exclusion on the Incidence of Yellow Vine Disease and of the Associated Bacterium in SquashBlake Bextine, Astri Wayadande, B D Bruton, et al.
Acta Physiologica (Oxford, England)|June 15, 2007
Activation of Ca(2+)-dependent protein kinase II during repeated contractions in single muscle fibres from mouse is dependent on the frequency of sarcoplasmic reticulum Ca(2+) releaseJ Aydin, T Korhonen, P Tavi, et al.
Plant Disease|March 3, 2019
Detection of Cucurbit Yellow Vine Disease in Squash and Pumpkin in MassachusettsR L Wick, J Lerner, S D Pair, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|May 16, 2023
Increased tetanic calcium in early fatigue of mammalian muscle fibers is accompanied by accelerated force development despite a decreased forceCecilia Leijding, Ida Viken, Joseph D Bruton, et al.
Journal of Cellular Physiology|January 30, 2010
Myogenic skeletal muscle satellite cells communicate by tunnelling nanotubesPasi Tavi, Topi Korhonen, Sandra L Hänninen, et al.
Phytopathology|October 24, 2008
Polymerase chain reaction detection and phylogenetic characterization of an agent associated with yellow vine disease of cucurbitsF J Avila, B D Bruton, J Fletcher, et al.
Phytopathology|October 24, 2008
Identification, Phylogenetic Analysis, and Biological Characterization of Serratia marcescens Strains Causing Cucurbit Yellow Vine DiseaseJ Rascoe, M Berg, U Melcher, et al.
Diabetes|July 28, 2005
Insulin and inositol 1,4,5-trisphosphate trigger abnormal cytosolic Ca2+ transients and reveal mitochondrial Ca2+ handling defects in cardiomyocytes of ob/ob miceJérémy Fauconnier, Johanna T Lanner, Shi-Jin Zhang, et al.
Cardiovascular Research|December 13, 2006
Insulin potentiates TRPC3-mediated cation currents in normal but not in insulin-resistant mouse cardiomyocytesJérémy Fauconnier, Johanna T Lanner, Ariane Sultan, et al.
The Journal of General Physiology|January 13, 2019
SR Ca<sup>2+</sup> leak in skeletal muscle fibers acts as an intracellular signal to increase fatigue resistanceNiklas Ivarsson, C Mikael Mattsson, Arthur J Cheng, et al.
Pageof 12