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Nature Genetics|November 4, 2000
Loss-of-function mutations in the EGF-CFC gene CFC1 are associated with human left-right laterality defectsR N Bamford, E Roessler, R D Burdine, et al.
Nature Communications|September 17, 2021
Bicc1 and Dicer regulate left-right patterning through post-transcriptional control of the Nodal inhibitor Dand5Markus Maerker, Maike Getwan, Megan E Dowdle, et al.
Journal of the American Society of Nephrology : JASN|February 29, 2008
SIX2 and BMP4 mutations associate with anomalous kidney developmentStefanie Weber, Jaclyn C Taylor, Paul Winyard, et al.
Biorxiv : the Preprint Server for Biology|July 19, 2024
Efficient Genome Editing with Chimeric Oligonucleotide-Directed EditingLong T Nguyen, Noah R Rakestraw, Brianna L M Pizzano, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 9, 2018
ZNRF3 functions in mammalian sex determination by inhibiting canonical WNT signalingAbigail Harris, Pam Siggers, Silvia Corrochano, et al.
Nature Genetics|December 7, 2010
The coiled-coil domain containing protein CCDC40 is essential for motile cilia function and left-right axis formationAnita Becker-Heck, Irene E Zohn, Noriko Okabe, et al.
Disease Models & Mechanisms|September 20, 2022
Promoting validation and cross-phylogenetic integration in model organism researchKeith C Cheng, Rebecca D Burdine, Mary E Dickinson, et al.
Nature Communications|April 12, 2026
Efficient genome editing with chimeric oligonucleotide-directed editingLong T Nguyen, Noah R Rakestraw, Brianna L M Pizzano, et al.
Nature Genetics|May 15, 2012
CCDC103 mutations cause primary ciliary dyskinesia by disrupting assembly of ciliary dynein armsJennifer R Panizzi, Anita Becker-Heck, Victoria H Castleman, et al.
American Journal of Human Genetics|September 6, 2014
CCDC151 mutations cause primary ciliary dyskinesia by disruption of the outer dynein arm docking complex formationRim Hjeij, Alexandros Onoufriadis, Christopher M Watson, et al.
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