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Organic Letters|July 31, 2012
Synthesis from D-altrose of (5R,6R,7R,8S)-5,7-dihydroxy-8-hydroxymethylconidine and 2,4-dideoxy-2,4-imino-D-glucitol, azetidine analogues of swainsonine and 1,4-dideoxy-1,4-imino-D-mannitolNoelia Araújo, Sarah F Jenkinson, R Fernando Martínez, et al.
Journal of Inherited Metabolic Disease|October 19, 2013
Non-specific accumulation of glycosphingolipids in GNE myopathyKatherine A Patzel, Tal Yardeni, Erell Le Poëc-Celic, et al.
Chembiochem : a European Journal of Chemical Biology|March 19, 2009
Synthesis and biological characterisation of novel N-alkyl-deoxynojirimycin alpha-glucosidase inhibitorsAmy J Rawlings, Hannah Lomas, Adam W Pilling, et al.
Chemistry (Weinheim an Der Bergstrasse, Germany)|June 9, 2021
Iminosugar C-Glycosides Work as Pharmacological Chaperones of NAGLU, a Glycosidase Involved in MPS IIIB Rare Disease*Sha Zhu, Yerri Jagadeesh, Anh Tuan Tran, et al.
Chemistry (Weinheim an Der Bergstrasse, Germany)|June 28, 2012
Scalable syntheses of both enantiomers of DNJNAc and DGJNAc from glucuronolactone: the effect of N-alkylation on hexosaminidase inhibitionAndreas F G Glawar, Daniel Best, Benjamin J Ayers, et al.
The Journal of Organic Chemistry|March 20, 2014
Nine of 16 stereoisomeric polyhydroxylated proline amides are potent β-N-acetylhexosaminidase inhibitorsBenjamin J Ayers, Andreas F G Glawar, R Fernando Martínez, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 6, 2012
Structural and mechanistic insight into N-glycan processing by endo-α-mannosidaseAndrew J Thompson, Rohan J Williams, Zalihe Hakki, et al.
Organic & Biomolecular Chemistry|October 4, 2014
N- and C-alkylation of seven-membered iminosugars generates potent glucocerebrosidase inhibitors and F508del-CFTR correctorsJ Désiré, M Mondon, N Fontelle, et al.
Antiviral Research|February 5, 2013
An iminosugar with potent inhibition of dengue virus infection in vivoStuart T Perry, Michael D Buck, Emily M Plummer, et al.
Nature Genetics|October 27, 2004
Infantile-onset symptomatic epilepsy syndrome caused by a homozygous loss-of-function mutation of GM3 synthaseMichael A Simpson, Harold Cross, Christos Proukakis, et al.
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